Liver cirrhosis, ascites, and hyperfibrinolysis [16]

American Journal of Gastroenterology

Volumn 96, Issue 11, 2001, Pages 3222-

  Piscaglia, Fabio ^a   Donati, Gabriele ^a   Giannini, Raffaele ^a   Bolondi, Luigi ^a  

^a UNIVERSITY OF BOLOGNA   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

PLASMINOGEN ACTIVATOR INHIBITOR;

ASCITES; BLOOD CLOTTING; CIRCADIAN RHYTHM; DISSEMINATED INTRAVASCULAR CLOTTING; ENDOTHELIUM CELL; ENDOTOXEMIA; FIBRINOLYSIS; HUMAN; INFECTION; INTESTINE ABSORPTION; LETTER; LIVER CIRRHOSIS; PATHOPHYSIOLOGY; PRIORITY JOURNAL;

EID: 0035191876     PISSN: 00029270     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1572-0241.2001.05298.x     Document Type: Letter

References (5)

1. Ascites fluid as a possible origin for hyperfibrinolysis in advanced liver disease
2. Coagulation abnormalities in liver cirrhosis. Hyperfibrinolysis resulting from clotting activation in patients with different degrees of cirrhosis
3. Association between low-grade disseminated intravascular coagulation and endotoxemia in patients with liver cirrhosis
4. Diurnal changes of fibrinolysis in patients with liver cirrhosis and esophageal varices
5. The pathogenesis of accelerated fibrinolysis in liver cirrhosis: A critical role for tissue plasminogen activator inhibitor