The irony of treating Wilson's disease

American Journal of Gastroenterology

Volumn 96, Issue 11, 2001, Pages 3055-3057

  Schilsky, Michael L ^a,b  

^a MOUNT SINAI MEDICAL CENTER   (United States)

^b MOUNT SINAI SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COPPER; IRON; PENICILLAMINE; ZINC;

CASE REPORT; DOSE RESPONSE; EDITORIAL; HUMAN; IRON BLOOD LEVEL; IRON OVERLOAD; LIVER BIOPSY; MALE; PATHOPHYSIOLOGY; PRIORITY JOURNAL; WILSON DISEASE;

EID: 0035185104     PISSN: 00029270     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0002-9270(01)03816-3     Document Type: Note

References (11)

1. A delicate balance: Homeostatic control of copper uptake and distribution
2. Iron accumulation in the liver of male patients with Wilson disease
3. Massive hepatic hemosiderosis in Wilson's disease
4. Discovery of the ceruloplasmin homologue hephaestin: New insight into the copper/iron connection
5. A novel duodenal iron-regulated transporter, IREG1, implicated in the basolateral transfer of iron to the circulation
6. Aceruloplasminemia: An inherited neurodegenerative disease with impairment of iron homeostasis
7. Prognosis of Wilsonian chronic active hepatitis
8. The use of trientine in preventing the effects of interrupting penicillamine therapy in Wilson's disease
9. Treatment of Wilson disease with ammonium tetrathiomolybdate. II. Initial therapy in 33 neurologically affected patients and follow-up with zinc therapy
10. Cytopenias secondary to copper depletion complicating ammonium tetrathiomolybdate therapy for Wilson's disease
11. Treatment of Wilson's disease with zinc: XV: Long-term follow-up studies