Increased dilator response to heptanol and octanol in aorta from DOCA-salt-hypertensive rats

Pharmacology

Volumn 62, Issue 1, 2001, Pages 29-35

  Leite, Romulo ^a,b   Webb, R Clinton ^b  

^a FEDERAL UNIVERSITY OF MINAS GERAIS   (Brazil)

^b UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

Author keywords

Calcium channels; Gap junctions; Heptanol, octanol; Hypertension, experimental, genetic; Muscle, smooth, vascular

Indexed keywords

CALCIUM CHANNEL L TYPE; DEOXYCORTICOSTERONE ACETATE; HEPTANOL; MINERALOCORTICOID; OCTANOL; PHENYLEPHRINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; AORTA CONSTRICTION; ARTICLE; BLOOD VESSEL REACTIVITY; CALCIUM CURRENT; CONTROLLED STUDY; GAP JUNCTION; HYPERTENSION; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; REGULATORY MECHANISM; SPONTANEOUSLY HYPERTENSIVE RAT; VASODILATATION;

ANIMALS; AORTA; DESOXYCORTICOSTERONE; DOSE-RESPONSE RELATIONSHIP, DRUG; HEPTANOL; HYPERTENSION; MALE; NIFEDIPINE; OCTANOLS; PHENYLEPHRINE; POTASSIUM CHLORIDE; RATS; RATS, INBRED SHR; RATS, INBRED WKY; RATS, SPRAGUE-DAWLEY; TETRADECANOYLPHORBOL ACETATE; VASOCONSTRICTION; VASOCONSTRICTOR AGENTS; VASODILATION; VASODILATOR AGENTS;

EID: 0035169108     PISSN: 00317012     EISSN: None     Source Type: Journal    
DOI: 10.1159/000056069     Document Type: Article

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28. +concentration