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Volumn 21, Issue 2, 2001, Pages 414-424
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Interleukin-6-induced STAT3 and AP-1 amplify hepatocyte nuclear factor 1-mediated transactivation of hepatic genes, an adaptive response to liver injury
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Author keywords
[No Author keywords available]
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Indexed keywords
INTERLEUKIN 6;
NUCLEAR FACTOR I;
PROTEIN SUBUNIT;
SOMATOMEDIN BINDING PROTEIN 1;
STAT3 PROTEIN;
TRANSCRIPTION FACTOR AP 1;
ANIMAL CELL;
ANIMAL TISSUE;
ARTICLE;
BINDING SITE;
CONTROLLED STUDY;
LIVER INJURY;
LIVER REGENERATION;
LIVER RESECTION;
MALE;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
TISSUE SPECIFICITY;
TRANSACTIVATION;
TRANSCRIPTION REGULATION;
ANIMALS;
DNA;
DNA-BINDING PROTEINS;
FIBRINOGEN;
GLUCOSE-6-PHOSPHATASE;
HEPATECTOMY;
HEPATOCYTE NUCLEAR FACTOR 1;
HEPATOCYTE NUCLEAR FACTOR 1-ALPHA;
HEPATOCYTE NUCLEAR FACTOR 1-BETA;
HUMANS;
INSULIN-LIKE GROWTH FACTOR BINDING PROTEIN 1;
INTERLEUKIN-6;
LIVER;
MALE;
MICE;
MICE, KNOCKOUT;
NUCLEAR PROTEINS;
PRECIPITIN TESTS;
PROMOTER REGIONS (GENETICS);
PROTEIN BINDING;
PROTO-ONCOGENE PROTEINS C-FOS;
PROTO-ONCOGENE PROTEINS C-JUN;
RESPONSE ELEMENTS;
RNA, MESSENGER;
STAT3 TRANSCRIPTION FACTOR;
TRANS-ACTIVATION (GENETICS);
TRANS-ACTIVATORS;
TRANSCRIPTION FACTOR AP-1;
TRANSCRIPTION FACTORS;
TUMOR CELLS, CULTURED;
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EID: 0035159293
PISSN: 02707306
EISSN: None
Source Type: Journal
DOI: 10.1128/MCB.21.2.414-424.2001 Document Type: Article |
Times cited : (120)
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References (65)
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