β Amyloid peptide (Aβ42) is internalized via the G-protein-coupled receptor FPRL1 and forms fibrillar aggregates in macrophages

FASEB Journal

Volumn 15, Issue 13, 2001, Pages 2454-2462

  Yazawa, Hiroshi ^a   Yu, Zu Xi ^b   Takeda, Kazuyo ^b   Le, Yingying ^a   Gong, Wanghua ^a   Ferrans, Victor J ^b   Oppenheim, Joost J ^a   Li, Chou Chi H ^a   Wang, Ji Ming ^a  

^a NATIONAL CANCER INSTITUTE   (United States)

^b NATIONAL HEART LUNG AND BLOOD INSTITUTE   (United States)

Author keywords

Alzheimer's disease; Amyloid ; Colocalization; Internalization

Indexed keywords

AMYLOID BETA PROTEIN; G PROTEIN COUPLED RECEPTOR; G PROTEIN COUPLED RECEPTOR FPRL1; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; ARTICLE; CHEMOTAXIS; COMPLEX FORMATION; CONFOCAL MICROSCOPY; CONTROLLED STUDY; DISEASE ASSOCIATION; HUMAN; HUMAN CELL; INFLAMMATION; INTERNALIZATION; MACROPHAGE; PRIORITY JOURNAL; PROTEIN DEGRADATION;

AMYLOID BETA-PROTEIN; APOPTOSIS; CELL LINE; CELLS, CULTURED; COLCHICINE; DOSE-RESPONSE RELATIONSHIP, DRUG; ENDOCYTOSIS; GTP-BINDING PROTEINS; HUMANS; MACROPHAGES; MICROSCOPY, CONFOCAL; OLIGOPEPTIDES; PEPTIDE FRAGMENTS; PROTEIN CONFORMATION; RECEPTORS, FORMYL PEPTIDE; RECEPTORS, IMMUNOLOGIC; RECEPTORS, LIPOXIN; RECEPTORS, PEPTIDE; TIME FACTORS;

EID: 0035158645     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.01-0251com     Document Type: Article

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