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Volumn 44, Issue 11, 2001, Pages 2642-2652
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Secondary necrosis is a source of proteolytically modified forms of specific intracellular autoantigens: Implications for systemic autoimmunity
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Author keywords
[No Author keywords available]
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Indexed keywords
AUTOANTIGEN;
ANTIGEN EXPRESSION;
ARTICLE;
AUTOIMMUNE DISEASE;
HUMAN;
HUMAN CELL;
NECROSIS;
PHAGOCYTOSIS;
PRIORITY JOURNAL;
PROTEIN DEGRADATION;
AMINO ACID CHLOROMETHYL KETONES;
ANTIBODIES, MONOCLONAL;
APOPTOSIS;
AUTOANTIGENS;
CASPASES;
DNA TOPOISOMERASES, TYPE I;
DRUG COMBINATIONS;
ETOPOSIDE;
HELA CELLS;
HUMANS;
JURKAT CELLS;
NECROSIS;
POLY(ADP-RIBOSE) POLYMERASES;
RIBONUCLEOPROTEIN, U1 SMALL NUCLEAR;
STAUROSPORINE;
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EID: 0035153242
PISSN: 00043591
EISSN: None
Source Type: Journal
DOI: 10.1002/1529-0131(200111)44:11<2642::AID-ART444>3.0.CO;2-8 Document Type: Article |
Times cited : (105)
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References (56)
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