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Volumn 33, Issue 2, 2001, Pages 439-447
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Pathogenesis of experimental neonatal woodchuck hepatitis virus infection: Chronicity as an outcome of infection is associated with a diminished acute hepatitis that is temporally deficient for the expression of interferon gamma and tumor necrosis factor-alpha messenger RNAs
a a b b c a a a |
Author keywords
[No Author keywords available]
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Indexed keywords
GAMMA INTERFERON;
MESSENGER RNA;
TUMOR NECROSIS FACTOR ALPHA;
ACUTE HEPATITIS;
ANIMAL TISSUE;
ARTICLE;
CELL TYPE;
DISEASE ASSOCIATION;
EFFECTOR CELL;
IMMUNE RESPONSE;
INFLAMMATORY CELL;
LIVER BIOPSY;
NONHUMAN;
OUTCOMES RESEARCH;
PATHOGENESIS;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
VIRUS CARRIER;
VIRUS LOAD;
WOODCHUCK;
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EID: 0035140877
PISSN: 02709139
EISSN: None
Source Type: Journal
DOI: 10.1053/jhep.2001.21748 Document Type: Article |
Times cited : (63)
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References (26)
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