Insulin fails to modulate the cardiac L-type Ca2+ current in type II diabetes patients - A possible link to cardiac dysfunction in diabetes mellitus [3]

Diabetologia

Volumn 44, Issue 2, 2001, Pages 269-

  Maier, S ^a   Lange, V ^a   Simm, A ^a   Walter, U ^a   Kirstein, M ^a  

^a UNIVERSITY OF WASHINGTON SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM CHANNEL L TYPE; INSULIN;

CALCIUM CURRENT; CARDIOMYOPATHY; ESSENTIAL HYPERTENSION; HEART DISEASE; HEART MUSCLE CELL; HEART VENTRICLE HYPERTROPHY; HUMAN; HUMAN CELL; INOTROPISM; LETTER; NON INSULIN DEPENDENT DIABETES MELLITUS; PRIORITY JOURNAL;

AGED; CALCIUM CHANNELS, L-TYPE; DIABETES MELLITUS, TYPE 2; HEART DISEASES; HUMANS; INSULIN; MIDDLE AGED;

EID: 0035131303     PISSN: 0012186X     EISSN: None     Source Type: Journal    
DOI: 10.1007/s001250051612     Document Type: Letter

References (5)

1. 2+ current in rat cardiac myocytes
2. 2+ current in human atrial myocytes by insulin
3. Membrane glycoprotein PC-1 and insulin resistance
4. Membrane glycoprotein PC-inhibition of insulin receptor function occurs via direct interaction with the receptor alpha-subunit
5. Insulin-glucose as adjunctive therapy for severe calcium channel antagonist poisoning