Atherosclerotic plaque rupture in the apolipoprotein E knockout mouse

Atherosclerosis

Volumn 154, Issue 2, 2001, Pages 399-406

  Johnson, Jason Lee ^a   Jackson, Christopher Langdale ^a  

^a UNIVERSITY OF BRISTOL   (United Kingdom)

Author keywords

Animal model; Atherosclerosis; Mouse; Plaque rupture

Indexed keywords

APOLIPOPROTEIN E; CHOLESTEROL; ELASTIN; LARD; LIPID;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATHEROSCLEROTIC PLAQUE; FAT INTAKE; FEMALE; IMMUNOCYTOCHEMISTRY; LIPID DIET; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; RUPTURE; THROMBUS;

ANIMALS; APOLIPOPROTEINS E; ARTERIOSCLEROSIS; BRACHIOCEPHALIC TRUNK; CHOLESTEROL; DIETARY FATS; FEMALE; MALE; MICE; MICE, KNOCKOUT; MUSCLE, SMOOTH, VASCULAR; RISK FACTORS; RUPTURE, SPONTANEOUS;

EID: 0035129613     PISSN: 00219150     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0021-9150(00)00515-3     Document Type: Article

References (10)

1. Why do plaques rupture?
2. The features of plaque disruption associated with thrombosis in the modified Constantinides animal model
3. Animal model that mimics atherosclerotic plaque rupture
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5. Diet-induced atherosclerosis in mice heterozygous and homozygous for apolipoprotein E gene disruption
6. Atherosclerosis in mice lacking apo E: Evaluation of lesional development and progression
7. ApoE-deficient mice develop lesions of all phases of atherosclerosis throughout the arterial tree
8. Dietary n-3 polyunsaturated fatty acids prevent the development of atherosclerotic lesions in mice: Modulation of macrophage secretory activities
9. Atherosclerosis susceptibility differences among progenitors of recombinant inbred strains of mice