Reduced heat shock proteins - A mechanism to explain higher cardiovascular events associated with doxazosin

Journal of Human Hypertension

Volumn 15, Issue 3, 2001, Pages 213-

  Hooper, P L ^a  

^a UNIVERSITY OF COLORADO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA ADRENERGIC RECEPTOR BLOCKING AGENT; CYTOKINE; DOXAZOSIN; HEAT SHOCK PROTEIN; HIGH DENSITY LIPOPROTEIN; ION CHANNEL; NORADRENALIN; PRAZOSIN; TRIACYLGLYCEROL;

APOPTOSIS; BRAIN INFARCTION; CARDIOVASCULAR DISEASE; CARDIOVASCULAR RISK; CLINICAL TRIAL; CYTOKINE RELEASE; HEART INFARCTION; HEART MUSCLE; HEART MUSCLE ISCHEMIA; HEART PROTECTION; HUMAN; LETTER; NONHUMAN; NORADRENALIN RELEASE; OXIDATION; PATHOPHYSIOLOGY; PROTEIN EXPRESSION; PROTEIN FOLDING;

EID: 0035127163     PISSN: 09509240     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.jhh.1001163     Document Type: Letter

References (8)

1. Do alpha blockers cause heart failure and stroke? Observations from ALLHAT
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4. Hemorheological and hemodynamic parameters in patients with essential hypertension and their modification by alpha-1 inhibitor drug treatment
5. Stress (heat shock) proteins - Molecular chaperones in cardiovascular biology and disease
6. Norepinephrine induces cardiac heat shock protein 70 and delayed cardioprotection in the rat through alpha 1 adrenoreceptors
7. Mice overexpressing rat heat shock protein 70 are protected against cerebral infarction
8. Effect of vasodilator therapy on mortality in chronic congestive heart failure. Results of a Veterans Administration Cooperative Study