Transition nuclear proteins during spermiogenesis: Unrepaired DNA breaks not allowed

Molecular Reproduction and Development

Volumn 58, Issue 4, 2001, Pages 357-358

  Kierszenbaum, Abraham L ^a,b  

^a CITY UNIVERSITY OF NEW YORK MEDICAL SCHOOL   (United States)

^b CITY UNIVERSITY OF NEW YORK   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

HISTONE; NUCLEAR PROTEIN;

ANIMAL CELL; ARTICLE; CELL FRACTIONATION; CHROMATIN CONDENSATION; DNA REPAIR; DNA STRAND BREAKAGE; FERTILIZATION; GENETIC ORGANIZATION; HAPLOIDY; NONHUMAN; NUCLEOSOME; PHASE TRANSITION; PRIORITY JOURNAL; REPORTER GENE; SPERMATOGENESIS; TISSUE SPECIFICITY; TRANSCRIPTION REGULATION;

ANIMALS; CHROMATIN; CHROMOSOMAL PROTEINS, NON-HISTONE; DNA DAMAGE; DNA REPAIR; DNA, SINGLE-STRANDED; GENE EXPRESSION REGULATION, DEVELOPMENTAL; INFERTILITY, MALE; MALE; MICE; MICE, KNOCKOUT; PROTAMINES; RATS; SPERMATOGENESIS;

EID: 0035119436     PISSN: 1040452X     EISSN: None     Source Type: Journal    
DOI: 10.1002/1098-2795(20010401)58:4<357::AID-MRD1>3.0.CO;2-T     Document Type: Article

References (3)

1. Mice with a targeted disruption of the H1t gene are fertile and undergo normal changes in structural chromosomal proteins during spermiogenesis
2. Nuclear morphogenesis during spermiogenesis
3. Abnormal spermatogenesis and reduced fertility in transition nuclear protein 1-deficiant mice