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Volumn 13, Issue 1, 2001, Pages 29-41
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Inactivation of JNK activity by mitogen-activated protein kinase phosphatase-2 in EAhy926 endothelial cells is dependent upon agonist-specific JNK translocation to the nucleus
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Author keywords
Endothelial cell; MAP kinase; MAP kinase phosphatase; N terminal c Jun kinase; p38 MAP kinase
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Indexed keywords
ENZYME ANTIBODY;
GLUTATHIONE TRANSFERASE;
HYBRID PROTEIN;
HYDROGEN PEROXIDE;
MITOGEN ACTIVATED PROTEIN KINASE INHIBITOR;
PHOSPHATASE;
TUMOR NECROSIS FACTOR ALPHA;
AMINO TERMINAL SEQUENCE;
ARTICLE;
CATALYSIS;
CELL CLONE;
CELL ISOLATION;
CELL NUCLEUS;
CELL STIMULATION;
CONTROLLED STUDY;
CYTOSOL;
DEPHOSPHORYLATION;
ENDOTHELIUM CELL;
ENZYME ACTIVITY;
ENZYME INACTIVATION;
GENETIC TRANSFECTION;
HUMAN;
HUMAN CELL;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
PROTEIN LOCALIZATION;
PROTEIN TRANSPORT;
REGULATORY MECHANISM;
SIGNAL TRANSDUCTION;
STIMULUS RESPONSE;
ANTIBODIES, ANTI-IDIOTYPIC;
CELL NUCLEUS;
ENDOTHELIUM;
HUMANS;
HYDROGEN PEROXIDE;
JNK MITOGEN-ACTIVATED PROTEIN KINASES;
MITOGEN-ACTIVATED PROTEIN KINASES;
P38 MITOGEN-ACTIVATED PROTEIN KINASES;
PHOSPHORYLATION;
PROTEIN TRANSPORT;
PROTEIN-TYROSINE-PHOSPHATASE;
SUBSTRATE SPECIFICITY;
TUMOR NECROSIS FACTOR-ALPHA;
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EID: 0035100127
PISSN: 08986568
EISSN: None
Source Type: Journal
DOI: 10.1016/S0898-6568(00)00121-2 Document Type: Article |
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Times cited : (30)
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References (33)
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