An intrinsic antibiotic mechanism in wounds and tissue-engineered skin [1]

Journal of Investigative Dermatology

Volumn 116, Issue 3, 2001, Pages 471-472

  Schmid, Peter ^a   Grenet, Olivier ^a   Medina, Jesús s ^a   Chibout, Salah Dine ^a   Osborne, Colin ^a   Cox, David A ^a  

^a NOVARTIS PHARMA AG   (Switzerland)

Author keywords

[No Author keywords available]

Indexed keywords

DEFENSIN;

EPIDERMIS; HUMAN; IMMUNOHISTOCHEMISTRY; LETTER; PRIORITY JOURNAL; PROTEIN EXPRESSION; SKIN INJURY; WOUND HEALING;

EID: 0035097737     PISSN: 0022202X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1523-1747.2001.01279.x     Document Type: Letter

References (8)

1. Rapid healing of venous ulcers and lack of clinical rejection with an allogeneic cultured human skin equivalent
2. A peptide antibiotic from human skin
3. Mucoid Pseudomonas aeruginosa, TNF-alpha, and IL-1beta, but not IL-6, induce human beta-defensin-2 in respiratory epithelia
4. Lipopolysaccharide induces physivcal proximity between CD14 and toll-like receptor 4 (TLR4) prior to nuclear translocation of NF-kappaB
5. Structure and mapping of the human beta-defensin HBD-2 gene and its expression at sites of inflammation
6. Molecules in focus: Human beta-defensin- 2
7. Development of a bilayered living skin construct for clinical application
8. β-defensins: Linking innate and adaptive immunity through dendritic and T cell CCR 6