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Volumn 18, Issue 3, 2001, Pages 237-253
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A mechanistic approach to modelling the risk of liver tumours in mice exposed to fumonisin B1 in the diet
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Author keywords
Compensatory proliferation; MVK model; Non genotoxic; Sphingolipid metabolism
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Indexed keywords
FUMONISIN B1;
SPHINGANINE;
SPHINGOLIPID;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BIOASSAY;
CANCER RISK;
CELL DEATH;
CELL PROLIFERATION;
CONTROLLED STUDY;
DOSE RESPONSE;
EXPOSURE;
FEMALE;
FOOD CONTAMINATION;
GENOTOXICITY;
LIPID METABOLISM;
LIVER CARCINOGENESIS;
LIVER TUMOR;
LIVER WEIGHT;
MALE;
MATHEMATICAL MODEL;
MOUSE;
MOUSE STRAIN;
MUTATION RATE;
NONHUMAN;
PREDICTION;
PRIORITY JOURNAL;
REPRODUCTION;
TARGET ORGAN;
TISSUE GROWTH;
TOXICOLOGY;
ANIMALS;
BODY WEIGHT;
CARBOXYLIC ACIDS;
CARCINOGENS, ENVIRONMENTAL;
CELL DEATH;
CELL DIVISION;
CELL TRANSFORMATION, NEOPLASTIC;
DOSE-RESPONSE RELATIONSHIP, DRUG;
FEMALE;
FOOD CONTAMINATION;
FUMONISINS;
LIVER;
LIVER NEOPLASMS, EXPERIMENTAL;
MALE;
MICE;
MICE, INBRED STRAINS;
MODELS, BIOLOGICAL;
MYCOTOXINS;
ORGAN SIZE;
RISK ASSESSMENT;
SEX FACTORS;
SPHINGOSINE;
ANIMALIA;
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EID: 0035062809
PISSN: 0265203X
EISSN: None
Source Type: Journal
DOI: 10.1080/02652030010021972 Document Type: Article |
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Times cited : (13)
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References (15)
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