Structural alterations in the translational attenuator of constitutively expressed erm(A) genes in Staphylococcus aureus [3]

Antimicrobial Agents and Chemotherapy

Volumn 45, Issue 5, 2001, Pages 1603-1604

  Schmitz, F J ^a   Petridou, J ^a   Astfalk, N ^a   Scheuring, S ^a   Kohrer K ^a   Verhoef, J ^a   Fluit, A C ^a   Schwarz, S ^a  

^a Bundesforschungsanst Landwirtschaft   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

LINCOSAMIDE; MACROLIDE; MIKAMYCIN B;

BINDING SITE; GENE DELETION; GENE EXPRESSION; GENE STRUCTURE; LETTER; MINIMUM INHIBITORY CONCENTRATION; NONHUMAN; OPEN READING FRAME; PHENOTYPE; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; R FACTOR; SEQUENCE ANALYSIS; STAPHYLOCOCCUS AUREUS; TRANSLATION REGULATION;

BACTERIAL PROTEINS; GENOME, BACTERIAL; HUMANS; METHYLTRANSFERASES; PROTEIN BIOSYNTHESIS; STAPHYLOCOCCUS AUREUS;

EID: 0035038131     PISSN: 00664804     EISSN: None     Source Type: Journal    
DOI: 10.1128/AAC.45.5.1603-1604.2001     Document Type: Letter

References (9)

1. Naturally occurring Staphylococcus epidermidis plasmid expressing constitutive macrolide-lincosamide-streptogramin B resistance contains a deleted attenuator
2. Bacterial resistance to macrolide, lincosamide, and streptogramin antibiotics by target modification
3. Nucleotide sequence of ermA, a macrolide-lincosamide-streptogramin B determinant in Staphylococcus aureus
4. Erythromycin-induced stabilization of ermA messenger RNA in Staphylococcus aureus and Bacillus subtilis
5. Prevalence of macrolide-resistance genes in Staphylococcus aureus and Enterococcus faecium isolates from 24 European university hospitals
6. Prevalence of resistance to MLS antibiotics in 20 European university hospitals participating in the European SENTRY surveillance program
7. Insights into erythromycin action from studies of its activity as inducer of resistance
8. Molecular analysis of the translational attenuator of a constitutively expressed erm(A) gene from Staphylococcusintermedius
9. Structural alterations in the translational attenuator of constitutively expressed ermC genes