Induction of pRb degradation by the human papillomavirus type 16 E7 protein is essential to efficiently overcome p16INK4a-imposed G1 cell cycle arrest

Journal of Virology

Volumn 75, Issue 10, 2001, Pages 4705-4712

  Giarre M ^a   Caldeira, S ^a   Malanchi, I ^a   Ciccolini, F ^a   Leaoo M J ^a   Tommasino, M ^a  

^a GERMAN CANCER RESEARCH CENTER DKFZ   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

GENE PRODUCT; PROTEIN E7; PROTEIN P16; RETINOBLASTOMA PROTEIN; UNCLASSIFIED DRUG; VIRUS PROTEIN;

ANIMAL CELL; ARTICLE; CELL CYCLE G1 PHASE; CELL TRANSFORMATION; CHIMERA; CONTROLLED STUDY; DNA FLANKING REGION; GENE ACTIVITY; GENE INDUCTION; GROWTH INHIBITION; NONHUMAN; ONCOGENE; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN MOTIF; PROTEIN STABILITY; SKIN DEFECT; TUMOR SUPPRESSOR GENE; WART VIRUS;

3T3 CELLS; ANIMALS; CELL DIVISION; CYCLIN-DEPENDENT KINASE INHIBITOR P16; G1 PHASE; HUMANS; MICE; ONCOGENE PROTEINS, VIRAL; PAPILLOMAVIRIDAE; RECOMBINANT FUSION PROTEINS; RETINOBLASTOMA PROTEIN;

EID: 0035032328     PISSN: 0022538X     EISSN: None     Source Type: Journal    
DOI: 10.1128/JVI.75.10.4705-4712.2001     Document Type: Article

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