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Volumn 13, Issue 8, 2001, Pages 1537-1546
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Contribution of intralaminar thalamic nuclei to spike-and-wave-discharges during spontaneous seizures in a genetic rat model of absence epilepsy
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Author keywords
Absence seizures; Centrolateral thalamic nucleus; GAERS; Microiontophoresis; Paracentral thalamic nucleus
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Indexed keywords
4 AMINOBUTYRIC ACID A RECEPTOR;
4 AMINOBUTYRIC ACID A RECEPTOR BLOCKING AGENT;
6,7 DINITRO 2,3 QUINOXALINEDIONE;
BICUCULLINE;
CGP 35347;
GLUTAMATE RECEPTOR ANTAGONIST;
UNCLASSIFIED DRUG;
ABSENCE;
ANIMAL CELL;
ANIMAL TISSUE;
ARTICLE;
ELECTROENCEPHALOGRAM;
GENETIC SUSCEPTIBILITY;
HISTOPATHOLOGY;
MALE;
NONHUMAN;
OSCILLATION;
PREFRONTAL CORTEX;
PRIORITY JOURNAL;
RAT;
SEIZURE;
SPIKE WAVE;
THALAMUS INTRALAMINAR NUCLEUS;
ACTION POTENTIALS;
ANIMALS;
BICUCULLINE;
ELECTROENCEPHALOGRAPHY;
ELECTROPHYSIOLOGY;
EPILEPSY, ABSENCE;
GABA ANTAGONISTS;
INTRALAMINAR THALAMIC NUCLEI;
MALE;
NEURONS;
RATS;
RATS, MUTANT STRAINS;
REACTION TIME;
RECEPTORS, GABA-A;
RECEPTORS, GABA-B;
RECRUITMENT, NEUROPHYSIOLOGICAL;
SEIZURES;
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EID: 0035003937
PISSN: 0953816X
EISSN: None
Source Type: Journal
DOI: 10.1046/j.0953-816x.2001.01537.x Document Type: Article |
Times cited : (61)
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References (41)
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