Overexpression of Hp95 induces G1 phase arrest in confluent HeLa cells

Differentiation

Volumn 67, Issue 4-5, 2001, Pages 139-153

  Wu, Y ^a   Pan, S ^a   Che, S ^a   He, G ^a   Nelman Gonzalez, M ^a   Weil, M M ^b   Kuang, J ^a  

^a UNIVERSITY OF TEXAS   (United States)

^b UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER   (United States)

Author keywords

Cell cycle; Hela cells; Oocyte maturation; Signal transduction; Xenopus laevis; Xp95

Indexed keywords

CYCLIN DEPENDENT KINASE; HEAT SHOCK PROTEIN; HEAT SHOCK PROTEIN 95; PROGESTERONE; UNCLASSIFIED DRUG;

ARTICLE; CELL CYCLE G1 PHASE; CELL GROWTH; CELL PROLIFERATION; CONTROLLED STUDY; ENZYME ACTIVITY; EXPRESSION VECTOR; GENE OVEREXPRESSION; GENETIC TRANSFECTION; HELA CELL; HUMAN; HUMAN CELL; MITOSIS INHIBITION; MOLECULAR CLONING; NONHUMAN; OOCYTE MATURATION; PRIORITY JOURNAL; SEQUENCE HOMOLOGY; SIGNAL TRANSDUCTION; TRANSGENE; XENOPUS LAEVIS;

EID: 0034988858     PISSN: 03014681     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1432-0436.2001.670406.x     Document Type: Article

References (54)

1. Gapped BLAST and PSI-BLAST: A new generation of protein database search programs
2. Flow cytometry and cell-separation procedures
3. Intestinal cell cycle regulations. Interactions of cyclin D1, Cdk4, and p21Cip1
4. Loss of wild-type p53 bestows a growth advantage on primary cortical astrocytes and facilitates their in vitro transformation
5. Malignant transformation of p53-deficient astrocytes is modulated by environmental cues in vitro
6. A new type of papillomavirus DNA, its presence in genital cancer biopsies and in cell lines derived from cervical cancer
7. A novel putative protein-tyrosine phosphatase contains a BRO1-like domain and suppresses Ha-ras-mediated transformation
8. Molecular cloning of a splice variant of Caenorhabditis elegans YNK1, a putative element in signal transduction
9. MPM-2 epitope sequence is not sufficient for recognition and phosphorylation by ME kinase-H
10. Identification and cloning of Xp95, a putative signal transduction protein in Xenopus oocytes
11. The glioma-associated protein SETA interacts with AIP1/Alix and ALG-2 and modulates apoptosis in astrocytes
12. Differences in the mechanisms of growth control in contact-inhibited and serum-deprived human fibroblasts
13. The human papilloma virus-16 E7 oncoprotein is able to bind to the retinoblastoma gene product
14. The regulation of E2F by pRB-family proteins
15. Xenopus oocyte maturation: New lessons from a good egg
16. Quantitation and experimental design
17. Protein-tyrosine kinases
18. The hallmarks of cancer
19. The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases
20. Regulation of cell proliferation by the E2F transcription factors
21. Contact inhibition-induced inactivation of the cyclin D-dependent kinase in rat fibroblast cell line, 3Y1
22. Induction of S phase by G1 regulatory factors
23. Compilation and analysis of sequences upstream from the translational start site in eukaryotic mRNAs
24. Interpreting cDNA sequences: Some insights from studies on translation
25. The ALG-2/AIP-complex, a modulator at the interface between cell proliferation and cell death? A hypothesis
26. S-Phase entry upon ectopic expression of G1 cyclin-dependent kinases in the absence of retinoblastoma protein phosphorylation
27. p27 is involved in N-cadherin-mediated contact inhibition of cell growth and S-phase entry
28. Recurring themes in oocyte maturation
29. Signalling networks that cause cancer
30. Alix, a novel mouse protein undergoing calcium-dependent interaction with the apoptotic-linked-gene (ALG-2) protein
31. Complex formation of human papillomavirus E7 proteins with the retinoblastoma tumor suppressor gene product
32. Characterization of the pH signal transduction pathway gene pa1A of Aspegillus nidulans and identification of possible homologs
33. BRO1, a novel gene that interacts with components of the pkc1p-mitogen-activated protein kinase pathway
34. Human cyclin E, a nuclear protein essential for the G1-to-S phase transition
35. Cellular transformation and the 'morphologic phenotype' of transformed cells
36. Human papillomavirus types 16 and 18 sequences in carcinoma cell lines of the cervix
37. p27Kip1, a cyclin-Cdk inhibitor, links transforming growth factor-beta and contact inhibition to cell cycle arrest
38. Structure and transcription of human papillomavirus sequences in cervical carcinoma cells
39. Anticancer drug targets: Cell cycle and checkpoint control
40. CDK inhibitors: Positive and negative regulators of G1-phase progression
41. Suppression of tumorigenicity in an aggressive cervical carcinoma induced by protein zero, a nervous system IgCAM
42. Kinases of the Src family: Structure and functions
43. Src and the control of cell division
44. p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21
45. Distinct roles for cyclin-dependent kinases in cell cycle control
46. Altered expression and function of E-cadherin in cervical intraepithelial neoplasia and invasive squamous cell carcinoma
47. 2+-binding protein ALG2 and Alzheimer's disease gene ALG-3
48. 2+-dependent reaction
49. Rapid analysis of gene expression (RAGE) facilitates universal expression profiling
50. p16INK4 mediates contact-inhibition of growth
51. Sodium butyrate blocks HeLa cells preferentially in early G1 phase of the cell cycle
52. Growth regulation by the E2F and DP transcription factor families
53. Opposite regulation of the expression of cyclin-dependent kinase inhibitors during contact inhibition
54. Structural basis for the binding of proline-rich peptides to SH3 domains