Caspase-3 activity and carbonyl cyanide m-chlorophenylhydrazone-induced apoptosis in HL-60 cells

Alternatives to Laboratory Animals

Volumn 29, Issue 3, 2001, Pages 243-249

  Mlejnek, P ^a  

^a MENDEL UNIVERSITY IN BRNO   (Czech Republic)

Author keywords

Caspase inhibitors; Caspase proteases; HL 60 cells; Necrosis; Nuclear apoptosis; Z DEVD FMK; Z VAD FMK

Indexed keywords

BENZYLOXYCARBONYLVALYLALANYLASPARTYL FLUOROMETHYL KETONE; CARBONYL CYANIDE CHLOROPHENYLHYDRAZONE; CASPASE 3; CELL DNA;

APOPTOSIS; CELL DEATH; CELL STRAIN HL 60; CHROMATIN CONDENSATION; CONCENTRATION RESPONSE; CONFERENCE PAPER; CONTROLLED STUDY; CYTOTOXICITY; DNA CLEAVAGE; ENZYME ACTIVITY; HUMAN; HUMAN CELL; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

EID: 0034978295     PISSN: 02611929     EISSN: None     Source Type: Journal    
DOI: 10.1177/026119290102900313     Document Type: Conference Paper

References (10)

1. The genetics of programmed cell death in the nematode Caenorhabiditis elegans
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5. 6-benzyladenosine
6. Effects of three epoxides - Ethylene oxide, propylene oxide and epichlorohydrin - On cell cycle progression and cell death in human diploid fibroblasts
7. The interaction of highly active uncouplers with mitochondria
8. Benzyloxycarbonyl-Val-Ala-Asp(OMe) fluoromethylketone (Z-VAD-FMK) inhibits apoptosis by blocking the processing of CPP32
9. Definition and incidence of apoptosis: An historical perspective
10. The apoptosis-necrosis paradox. Apoptotic proteases activated after mitochondrial permeability transition determine the mode of cell death