Cellular responses by exposure to heavy-ions

Advances in Space Research

Volumn 27, Issue 2, 2001, Pages 401-409

  Morimoto, S ^a   Gordon, A ^a   Fukunishi, N ^a   Yatagai, F ^a  

^a INST OF PHYS AND CHEMICAL RESEARCH   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BIOCHEMISTRY; HEAVY IONS; IRON; IRRADIATION; PROTEINS; RADIATION EFFECTS; X RAYS;

LOW-DOSE EFFECTS;

CELLS;

RADIATION EXPOSURE;

EID: 0034974271     PISSN: 02731177     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0273-1177(01)00008-4     Document Type: Article

References (13)

1. Different cytotoxic and mutagenic responses induced by X-rays in two human lymphoblastoid cell lines derived from a single donor
2. DNA double-strand break rejoining deficiency in TK6 and other human B- lymphoblast cell lines
3. Effects of low dose irradiation on TK6 and U937 cells: Induction of p53 and its role in cell-cycle delay and the adaptive response
4. Radioadaptive response: Efficient repair of radiation-induced DNA damage in adapted cells
5. Complex hprt deletion events are recovered after exposure of human lymphoblastoid cells to high-LET carbon and neon ion beams
6. Analysis of mutations in the human HPRT gene induced by accelerated heavy-ion irradiation
7. Adaptive response of human cultured cells to heavy-ion irradiation
8. Low-dose-rate radiation attenuates the response of the tumor suppressor TP53
9. On the reaction kinetics of the radioadaptive response in cultured mouse cells
10. Coordinated regulation of radioadaptive response by protein kinase C and p38 mitogen-activated protein kinase
11. Aspects of the adaptive responses to very low doses of radiation and other agents
12. Different capacities for recombination in closely related human lymphoblastoid cell lines with different mutational responses to X-irradiation
13. The relative radiosensitivity of TK6 and WI-L2-NS lymphoblastoid cells derived from a common source is primarily determined by their p53 mutational status