Ion channel dysfunction and peripheral nerve disorders;Dysfonction des canaux ioniques et neuropathies périphériques

Revue Neurologique

Volumn 157, Issue 5, 2001, Pages 477-479

  Lefaucheur, J P ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

CARBAMAZEPINE; CIGUATOXIN; GABAPENTIN; ION CHANNEL; TETRODOTOXIN;

DEMYELINATION; DEPOLARIZATION; EDITORIAL; ELECTROPHYSIOLOGY; GUILLAIN BARRE SYNDROME; HYPERPOLARIZATION; ION TRANSPORT; NERVE CONDUCTION; NEUROPATHY; PATHOPHYSIOLOGY; PERIPHERAL NEUROPATHY; POTASSIUM CONDUCTANCE;

HUMANS; ION CHANNELS; NERVE FIBERS; NEURAL CONDUCTION; PERIPHERAL NERVOUS SYSTEM DISEASES; POTASSIUM CHANNELS; SODIUM CHANNELS;

EID: 0034967728     PISSN: 00353787     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Editorial

References (20)

1. Clinical observations on 3009 cases of ciguatera (fish poisoning) in the South Pacific
2. Axonal ion channel dysfunction in amyotrophic lateral sclerosis
3. Na+ channel immunolocalization in peripheral mammalian axons and changes following nerve injury and neuroma formation
4. Increased numbers of sodium channels form along demyelinated axons
5. Sodium channel accumulation in humans with painful neuromas
6. The physiological effect of anti-GM1 antibodies on saltatory conduction and transmembrane currents in single motor axons
7. Activity-dependent conduction block in multifocal motor neuropathy
8. Conduction block in carpal tunnel syndrome
9. Excitability properties of median and peroneal motor axons
10. Recovery of excitability of cutaneous afferents in the median and sural nerves following activity
11. Paralytic shellfish poisoning: A case report and serial electrophysiologic observations
12. The pathological and clinical dynamics of multiple sclerosis
13. Strength-duration properties of sensory and motor axons in amyotrophic lateral sclerosis
14. Mechanisms of paresthesias arising from healthy axons
15. Nerve conduction study of human tetrodotoxication
16. Erythrocyte Na/K ATPase activity, metabolic control and neuropathy in insulin dependent diabetic patients
17. Acquired neuromyotonia: Evidence for autoantibodies directed against K+ channels of peripheral nerves
18. Association of diabetic neuropathy with Na/K ATPase gene polymorphism
19. Sodium channels, excitability of primary sensory neurons, and the molecular basis of pain
20. Conduction slowing without conduction block of compound muscle and nerve action potential due to channel block