Clearing the way to mechanisms of autoimmunity

Nature Medicine

Volumn 7, Issue 6, 2001, Pages 664-665

  Rosen, A ^a   Casciola Rosen, L ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIBODY; DNA ANTIBODY; MER TYROSINE KINASE; PROTEIN TYROSINE KINASE; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANTIBODY TITER; APOPTOSIS; AUTOIMMUNITY; CLEARANCE; CONTROLLED STUDY; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; SHORT SURVEY; THYMOCYTE;

ANIMALS; APOPTOSIS; AUTOANTIGENS; AUTOIMMUNE DISEASES; HUMANS; MACROPHAGES; MICE; NEURAL CELL ADHESION MOLECULES; PROTO-ONCOGENE PROTEINS; RECEPTOR PROTEIN-TYROSINE KINASES; RECEPTORS, CELL SURFACE;

ANIMALIA;

EID: 0034957380     PISSN: 10788956     EISSN: None     Source Type: Journal    
DOI: 10.1038/89034     Document Type: Short Survey

References (10)

1. Autoantigens targeted in systemic lupus erythematosus are clustered in two populations of surface structures on apoptotic keratinocytes
2. Natural adjuvants: Endogenous activators of dendritic cells
3. Consequences of cell death: Exposure to necrotic tumor cells, but not primary tissue cells or apoptotic cells, induces the maturation of immunostimulatory dendritic cells
4. Phagocytosis and clearance of apoptotic cells is mediated by Mer
5. Corpse clearance defines the meaning of cell death
6. Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies
7. The lupus paradox
8. Serum amyloid P component controls chromatin degradation and prevents antinuclear autoimmunity
9. A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo
10. Apoptotic cell clearance in systemic lupus erythematosus - I. Opsonization by antiphospholipid antibodies