Walker tumor cells express larger amounts of the antiapoptotic protein Bcl-2 and presents higher resistance to toxic concentrations of Ca2+ than the tumor cells K 562

Drug Development Research

Volumn 52, Issue 4, 2001, Pages 508-514

  Milani, Graziela ^a   Kowaltowski, Alicia J ^a,b   Saad, Sara T O ^a   Metze, Konradin ^a   Vercesi, Anibal E ^a  

^a UNIVERSITY OF CAMPINAS   (Brazil)

^b UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

Bcl 2; Calcium; Mitochondria; Tumor

Indexed keywords

CALCIUM; DIGITONIN; FURA 2; PROTEIN BCL 2;

ANIMAL CELL; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CELL MEMBRANE PERMEABILITY; CELL ORGANELLE; CONTROLLED STUDY; ELECTRON MICROSCOPY; FLUORESCENCE; GENE EXPRESSION; GENE OVEREXPRESSION; GENETIC TRANSFECTION; IMMUNOCYTOCHEMISTRY; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; NONHUMAN; WALKER CARCINOMA;

EID: 0034947325     PISSN: 02724391     EISSN: None     Source Type: Journal    
DOI: 10.1002/ddr.1154     Document Type: Article

References (3)

1. Åkerman KEO, Wikstron MKF. 1976. Safranine as a probe for mitochondrial membrane potential. FEBS Lett 68:191-197.
2. Andreyev AY, Fahy B, Fiskum G. 1998. Cytochrome c release from brain mitochondria is independent of the mitochondrial permeability transition. FEBS Lett 439:373-376.
3. Cadenas E, Boveris A. 1980. Enhancement of hydrogen peroxide formation by protophores and ionophores in antimycin-supplemented mitochondria. Biochem J 188:31-37.