Elevated interleukin-18 levels in the urine of nephrotic patients

Nephron

Volumn 88, Issue 4, 2001, Pages 334-339

  Matsumoto, Koichi ^a   Kanmatsuse, Katsuo ^b  

^a SAITAMA PREFECTURAL UNIVERSITY   (Japan)

^b NIHON UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

IgA nephropathy; Interleukin 18; Minimal change nephritic syndrome; Nephrotic syndrome

Indexed keywords

INTERLEUKIN 18; PREDNISOLONE;

ADULT; ARTICLE; CLINICAL ARTICLE; CONTROLLED STUDY; DISEASE ACTIVITY; ENZYME LINKED IMMUNOSORBENT ASSAY; FEMALE; HELPER CELL; HUMAN; IMMUNOGLOBULIN A NEPHROPATHY; MALE; MINIMAL CHANGE GLOMERULONEPHRITIS; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEINURIA; STEROID THERAPY; URINE LEVEL;

ADULT; COMPLEMENT SYSTEM PROTEINS; FEMALE; GLOMERULONEPHRITIS, IGA; HUMANS; IMMUNOGLOBULIN A; INTERLEUKIN-18; KIDNEY GLOMERULUS; MALE; NEPHROTIC SYNDROME; PREDNISOLONE; PROTEINURIA;

EID: 0034939382     PISSN: 00282766     EISSN: None     Source Type: Journal    
DOI: 10.1159/000046017     Document Type: Article

References (14)

1. T-lymphocyte populations, cytokines and other growth factors in serum and urine of children with idiopathic nephritic syndrome
2. Th1 and Th2 cytokine mRNA profiles in childhood nephritic syndrome: Evidence for increased IL-13 mRNA expression in relapse
3. Interleukin-15 and interleukin-12 have an addictive effect on the release of vascular permeability factor by peripheral blood mononuclear cells in normal subjects and in patients with minimal-change nephritis syndrome
4. Th1 and Th2 helper cell subsets affect patterns of injury and outcomes in glomerulonephritis
5. Interleukin-18 and interleukin-1β: Two cytokine substrates for ICE (Caspase-1)
6. Interleukin-18, rheumatoid arthritis, and tissue destruction
7. A retrospective analysis of the natural history of primary IgA nephropathy worldwide
8. IL-12 upregulates IL-18 receptor expression on T cells, Th1 cells, and B cells: Synergism with IL-18 for IFN-γ production
9. Interleukin-18 and interleukin-12 synergize to stimulate the production of vascular permeability factor by T lymphocytes in normal subjects and in patients with minimal-change nephritic syndrome
10. Differentiation between vascular permeability factor and IL-2 in lymphocyte supernatants from patients with minimal-change nephritic syndrome
11. Interleukin-18 binding protein: A novel modulator of the Th1 cytokine response
12. Structural requirements of six naturally occurring isoforms of the IL-18 binding protein to inhibit IL-18
13. Anti-inflammatory actions of steroids: Molecular mechanism