Role of caspases in neuronal apoptosis

Drug Development Research

Volumn 52, Issue 4, 2001, Pages 515-533

  Ventimiglia, Roseann ^a,b   Lau, Lit Fui ^a,b   Kinloch, Ross A ^a,b   Hopkins, Andrew ^a,b   Karran, Eric H ^a,b   Petalidis, Lawrence P ^a,b   Ward, Robin V ^a,b  

^a PFIZER GLOBAL RESEARCH AND DEVELOPMENT   (United Kingdom)

^b PFIZER GLOBAL RESEARCH AND DEVELOPMENT   (United States)

Author keywords

Apoptosis; Cysteine protease; Neurodegeneration; Programmed cell death

Indexed keywords

CASPASE; CASPASE INHIBITOR; CYSTEINE PROTEINASE; INTERLEUKIN 1BETA CONVERTING ENZYME INHIBITOR; ISATIN DERIVATIVE; PRALNACASAN; UNCLASSIFIED DRUG;

APOPTOSIS; BIOTECHNOLOGY; CONTROLLED STUDY; DEGENERATIVE DISEASE; DRUG STRUCTURE; DRUG TARGETING; HOMEOSTASIS; HUMAN; HUMAN CELL; MOLECULAR RECOGNITION; NERVE CELL; REVIEW; SIGNAL TRANSDUCTION;

EID: 0034938125     PISSN: 02724391     EISSN: None     Source Type: Journal    
DOI: 10.1002/ddr.1155     Document Type: Review

References (1)

1. Antonsson B, Montessuit S, Lauper S, Eskes R, Martinou JC. 2000. Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria. Biochem J 345:271-278.