Dormancy and persistence in chronic infection: Role of the general stress response in resistance to chemotherapy [1]

Journal of Antimicrobial Chemotherapy

Volumn 48, Issue 1, 2001, Pages 141-142

  Brown, M R W ^a   Smith, A W ^a  

^a University of Bath   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIINFECTIVE AGENT; BIOCIDE; MESSENGER RNA; SIGMA FACTOR; SIGMA FACTOR RPOS; UNCLASSIFIED DRUG;

ANTIBIOTIC RESISTANCE; BIOFILM; BIOMASS; CELL DIVISION; CELL SURVIVAL; CYSTIC FIBROSIS; EUKARYOTE; HUMAN; HYDROPHOBICITY; INFECTION; LETTER; MACROPHAGE; MICELLE; MYCOBACTERIUM TUBERCULOSIS; NONHUMAN; PATHOGENICITY; PHAGOCYTE; PHENOTYPE; PROTOZOON; PSEUDOMONAS AERUGINOSA; STRESS;

BACTERIA; BIOFILMS; CHRONIC DISEASE; DRUG RESISTANCE, MICROBIAL;

EID: 0034926378     PISSN: 03057453     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Letter

References (8)

1. Dormant tubercle bacilli: The key to more effective TB chemotherapy?
2. Unexplored reservoirs of pathogenic bacteria: Protozoa and biofilms
3. Interplay of global regulators and cell physiology in the general stress response of Escherichia coli
4. Quorum-sensing signals indicate that cystic fibrosis lungs are infected with bacterial biofilms
5. Regulation of quorum sensing by RpoS in Pseudomonas aeruginosa
6. General stress response master regulator rpoS is expressed in human infection: A possible role in chronicity
7. Intracellular growth in Acanthamoeba castellani affects monocyte entry mechanisms and enhances virulence of Legionella pneumophila
8. Environmental and clinical isolates of Pseudomonas aeruginosa show pathogenic and biodegradative properties irrespective of their origin