Unusual halo nevi - Darkening rather than lightening of the central nevus

Dermatology

Volumn 202, Issue 4, 2001, Pages 324-327

  Huynh, Phung M ^a   Lazova, Rossitza ^a   Bolognia, Jean L ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Darkening; Halo nevus; Perifollicular hypopigmentation

Indexed keywords

ADULT; ARTICLE; CASE REPORT; DISEASE CLASSIFICATION; HISTOLOGY; HUMAN; HUMAN TISSUE; HYPERPIGMENTATION; LYMPHOCYTE; MALE; NEVUS; PRIORITY JOURNAL; SKIN PIGMENTATION;

ADOLESCENT; BACK; HUMANS; HYPERPIGMENTATION; MALE; NEVUS, PIGMENTED; SKIN NEOPLASMS;

EID: 0034917330     PISSN: 10188665     EISSN: None     Source Type: Journal    
DOI: 10.1159/000051666     Document Type: Article

References (22)

1. The halo nevus
2. Consensus Development Panel on Early Melanoma: Diagnosis and treatment of early melanoma
3. Natural regression of the melanocytic nevus
4. Broad spectrum of leukoderma acquisitum centrifugum
5. Halo nevi
6. Halo nevus with exceptional clinical features: Apparent arrest of nevus cell degeneration and return of pigment to halo
7. Halo nevus
8. Benign neoplasias and hyperplasias of melanocytes
9. Benign melanocytic neoplasms: Congenital and acquired nevi
10. Perifollicular hypopigmentation: A cause of variegate pigmentation and irregular border in melanocytic nevi
11. Clinical and histopathological studies on spotted grouped pigmented nevi with special reference to eccrine-centered nevus
12. Characterization of the mononuclear infiltrate involved in regression of halo nevi
13. CD69 expression and tumor necrosis factor α immunoreactivity in the inflammatory cell infiltrate of halo naevi
14. Presence of activated lymphocytes in the peripheral blood of patients with halo nevi
15. Presence of T cells and macrophages in inflammatory vitiligo skin parallels melanocyte disappearance
16. High frequency of skin-homing melanocyte-specific cytotoxic T lymphocytes in autoimmune vitiligo
17. 2 but not MSH stimulate the proliferation of pigment cells in the pinnal epidermis of the DBA/2 mouse
18. Postinflammatory hypopigmentation and hyperpigmentation