High prevalence of factor V leiden mutation among healthy individuals and patients with deep venous thrombosis in Lebanon: Is the eastern Mediterranean region the area of origin of this mutation?

Thrombosis and Haemostasis

Volumn 86, Issue 2, 2001, Pages 723-724

  Taher, A ^a   Khalil, I ^a   Shamseddine, A ^a   El Ahdab, F ^a   Bazarbachi, A ^a  

^a AMERICAN UNIVERSITY OF BEIRUT   (Lebanon)

Author keywords

[No Author keywords available]

Indexed keywords

BLOOD CLOTTING FACTOR 5 LEIDEN; TYPE II SITE SPECIFIC DEOXYRIBONUCLEASE;

ASIAN; CLINICAL ARTICLE; CONTROLLED STUDY; DEEP VEIN THROMBOSIS; DNA CLEAVAGE; GENE MUTATION; GENETIC ASSOCIATION; HETEROZYGOSITY; HOMOZYGOSITY; HUMAN; LEBANON; LETTER; MAJOR CLINICAL STUDY; MONONUCLEAR CELL; NEGRO; POPULATION RESEARCH; PREVALENCE; PRIORITY JOURNAL; REGIONALIZATION; RISK FACTOR;

EID: 0034898551     PISSN: 03406245     EISSN: None     Source Type: Journal    
DOI: 10.1055/s-0037-1616118     Document Type: Letter

References (7)

1. World distribution of Factor V Leiden
2. Epidemiology of Factor V Leiden: Clinical implications
3. Mutation in the gene coding for coagulation Factor V and the risk of myocardial infarction, stroke, and venous thrombosis in apparently healthy men
4. Anticoagulant protein C pathway defective in a majority of thrombophilic patients
5. Venous thrombosis due to poor response to activated protein C: Leiden thrombophilia study
6. Resistance to activated protein C as a basis for venous thrombosis
7. The prevalence of Factor V R506Q mutation-Leiden among apparently health Lebanese