Histamine inhibits chemotaxis, phagocytosis, superoxide anion production, and the production of TNFα and IL-12 by macrophages via H2-receptors

International Immunopharmacology

Volumn 1, Issue 9-10, 2001, Pages 1867-1875

  Azuma, Yasutaka ^a   Shinohara, Mitsuko ^a   Wang, Pao Li ^a   Hidaka, Atsushi ^a   Ohura, Kiyoshi ^a  

^a OSAKA DENTAL UNIVERSITY   (Japan)

Author keywords

H2 histamine receptors; Histamine; IL 12; Macrophage; Phagocytosis; TNF

Indexed keywords

DIMAPRIT; HISTAMINE; INTERLEUKIN 12; LIPOPOLYSACCHARIDE; PHORBOL 13 ACETATE 12 MYRISTATE; SUPEROXIDE; TUMOR NECROSIS FACTOR ALPHA; ZYMOSAN;

ANIMAL CELL; ARTICLE; CHEMOTAXIS; CONCENTRATION RESPONSE; CONTROLLED STUDY; CYTOKINE PRODUCTION; DRUG EFFECT; DRUG INHIBITION; ESCHERICHIA COLI; IN VITRO STUDY; MACROPHAGE; MALE; NONHUMAN; OPSONIZATION; PHAGOCYTOSIS; PRIORITY JOURNAL; RAT;

ANIMALS; CHEMOTAXIS, LEUKOCYTE; CYTOKINES; DIMAPRIT; HISTAMINE; HISTAMINE AGONISTS; IMMUNITY, CELLULAR; INTERLEUKIN-12; MACROPHAGES, PERITONEAL; MALE; MICE; PHAGOCYTOSIS; RATS; RATS, WISTAR; RECEPTORS, HISTAMINE H2; SUPEROXIDES; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0034881366     PISSN: 15675769     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1567-5769(01)00112-6     Document Type: Article

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