bcl2, p53 and bax in thyroid tumors and their relation to apoptosis

Neoplasma

Volumn 48, Issue 4, 2001, Pages 299-301

  Farid, P ^a   Gomba, S Z ^a   Peter I ^a   Szende, B ^a  

^a SEMMELWEIS UNIVERSITY   (Hungary)

Author keywords

Bax; p53; Thyroid tumors

Indexed keywords

PROTEIN BAX; PROTEIN BCL 2; PROTEIN P53;

ADULT; AGED; APOPTOSIS; ARTICLE; CLINICAL ARTICLE; HEAD AND NECK TUMOR; HUMAN; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; PROTEIN EXPRESSION; THYROID ADENOMA; THYROID CARCINOMA; THYROID TUMOR;

EID: 0034876933     PISSN: 00282685     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (12)

1. 2 and p53 protein expression
2. Expression of the cell death-inducing gene bax in carcinomas developed from the follicular cells of the thyroid gland
3. Detection of DNA strand breaks in individual apoptotic cells by the in situ terminal deoxynucleotidyl transferase and nick translation assay
4. 2 and bax expression in thyroid tumors
5. 2 in oncocytic adenomas and carcinomas of the thyroid gland
6. 2 heterodimerizes in vivo with a conserved homolog, bax that accelerates programmed cell death
7. 2 protein expression in carcinomas originating from the follicular epithelium of the thyroid gland
8. 2 and the regulation of programmed cell death
9. Mechanism of apoptosis: Integration of genetic, biochemical and cellular indication
10. Pathology of human disease
11. Apoptosis: Cell death in tissue regulation