Is stagnating flow in former feeding arteries an indication of cerebral hypoperfusion after resection of arteriovenous malformations?

Journal of Neurosurgery

Volumn 95, Issue 1, 2001, Pages 36-43

  Meyer, B ^a   Urbach, H ^a   Schaller, C ^a   Schramm, J ^a  

^a UNIVERSITY OF BONN   (Germany)

Author keywords

Cerebral arteriovenous malformation; Cerebral oxygen saturation; Cerebrovascular circulation; Stagnating flow

Indexed keywords

ADOLESCENT; ADULT; AGED; ARTERIOVENOUS MALFORMATION; ARTERY BLOOD FLOW; ARTICLE; BRAIN ANGIOGRAPHY; BRAIN BLOOD FLOW; BRAIN EDEMA; BRAIN HEMORRHAGE; BRAIN ISCHEMIA; BRAIN PERFUSION; CHILD; FEMALE; HUMAN; MAJOR CLINICAL STUDY; MALE; OXYGEN SATURATION; POSTOPERATIVE PERIOD; PRIORITY JOURNAL;

EID: 0034871194     PISSN: 00223085     EISSN: None     Source Type: Journal    
DOI: 10.3171/jns.2001.95.1.0036     Document Type: Article

References (34)

1. Occlusive hyperemia: A theory for the hemodynamic complications following resection of intracerebral arteriovenous malformations
2. Hypertensive encephalopathy: A warning
3. Lack of correlation between pattern of collateralization and misery perfusion in patients with carotid occlusion
4. Evidence against cerebral vasospasm during acutely induced hypertension
5. The Erlangen micro-lightguide spectrophotometer EMPHO I
6. Cerebral sinus and venous thrombosis in rats induces long-term deficits in brain function and morphology-evidence for a cytotoxic genesis
7. Appraisal of the angiographic circulation time as an index of cerebral blood flow
8. Experimental study of venous circulatory disturbance by dural sinus occlusion
9. Vasomotion induces regular major oscillations in jejunal mucosal tissue oxygenation
10. Results and value of immediate postoperative angiography after operations for arteriovenous malformations
11. Cerebral hemodynamics in angioma patients: An intraoperative study
12. Hypertensive encephalopathy. Magnetic resonance imaging demonstration of reversible cortical and white matter lesions
13. Normal perfusion pressure breakthrough syndrome in giant arteriovenous malformations
14. Effects of acutely induced hypertension in cats on pial arteriolar caliber, local cerebral blood flow, and the blood-brain barrier
15. Distributions of local oxygen saturation and its response to changes of mean arterial blood pressure in the cerebral cortex adjacent to arteriovenous malformations
16. Capillary oxygen saturation and tissue oxygen pressure in the rat cortex at different stages of hypoxic hypoxia
17. Pathophysiologic assessment of stagnating arteries after removal of arteriovenous malformations
18. Retrograde thrombosis of feeding arteries after removal of arteriovenous malformations
19. Complications of surgery for arteriovenous malformations of the brain
20. Delayed neurological deterioration following resection of arteriovenous malformations of the brain
21. A study of the reactivity of feeding vessels to arteriovenous malformations: Correlation with clinical outcome
22. Alterations of regional cerebral blood flow and oxygen saturation in a rat sinus-vein thrombosis model
23. Local cerebral blood flow in a rat cortical vein occlusion model
24. Hemodynamic aspects of cerebral arteriovenous malformations
25. Management of residual dysplastic vessels after cerebral arteriovenous malformation resection: Implications for postoperative angiography
26. Blood pressure monitoring in feeding arteries of cerebral arteriovenous malformations during embolization: A preventive role in hemodynamic complications
27. A proposed grading system for arteriovenous malformations
28. Surgical management of large AVM's by staged embolization and operative excision
29. Normal perfusion pressure breakthrough theory
30. Blood vessel changes in chronic experimental arteriovenous fistulas
31. Perinidal hypervascular network on immediate postoperative angiogram after removal of large arteriovenous malformations located distant from the arterial circle of Willis
32. Cerebral blood flow alterations in a rat model of cerebral sinus thrombosis
33. Cerebral arteriovenous malformations: Flow quantitation by means of two-dimensional cardiac-gated phase-contrast MR imaging
34. Occlusive hyperemia: A new way to think about an old problem