Decreased neuronal excitability in hippocampal neurons of mice exposed to cyclic hypoxia

Journal of Applied Physiology

Volumn 91, Issue 3, 2001, Pages 1245-1250

  Gu, Xiang Q ^a   Haddad, Gabriel G ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Excitability; Oxygen deprivation; Sodium channels

Indexed keywords

SODIUM CHANNEL; SODIUM ION;

ANIMAL CELL; ARTICLE; CONTROLLED STUDY; HIPPOCAMPUS; HYPOXIA; MEMBRANE ELECTROPHYSIOLOGY; MOUSE; NERVE CELL CULTURE; NERVE CELL EXCITABILITY; NERVE POTENTIAL; NONHUMAN; PRIORITY JOURNAL; SODIUM CURRENT;

EID: 0034870755     PISSN: 87507587     EISSN: None     Source Type: Journal    
DOI: 10.1152/jappl.2001.91.3.1245     Document Type: Article

References (10)

1. Sleep apnea, hypopnea and oxygen desaturation in normal subjects. A strong male predominance
2. Functional properties of rat and human neocortical voltage-sensitive sodium currents
3. A defect in skeletal muscle sodium channel deactivation exacerbates hyperexcitability in human paramyotonia congenita
4. 2 deprivation in the central nervous system: On mechanisms of neuronal response, differential sensitivity and injury
5. Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patches
6. + channels must deactivate to recover from inactivation
7. + channels
8. + current in rat hippocampal neurones via protein kinase C
9. Chronic hypoxia in vivo renders neocortical neurons more vulnerable to subsequent acute hypoxic stress
10. The development of the brain and perinatal hypoxia