The time course of natriuretic hormones as plasma markers of myocardial recovery in heart transplant candidates during ventricular assist device support reveals differences among device types

Journal of Heart and Lung Transplantation

Volumn 20, Issue 9, 2001, Pages 949-955

  Milting, Hendrik ^a   El Banayosy, Aly ^a   Kassner, Astrid ^a   Fey, Oliver ^a   Sarnowski, Peter ^a   Arusoglu, Latif ^a   Thieleczek, Rolf ^b   Brinkmann, Thomas ^a   Kleesiek, Knut ^a   Körfer, Reiner ^a  

^a RUHR UNIVERSITY BOCHUM   (Germany)

^b RUHR UNIVERSITY BOCHUM   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

ATRIAL NATRIURETIC FACTOR; BIOLOGICAL MARKER; BRAIN NATRIURETIC PEPTIDE; NATRIURETIC FACTOR;

ADULT; AGED; ARTICLE; ASSISTED CIRCULATION; CARDIOMYOPATHY; CLINICAL ARTICLE; CONTROLLED STUDY; DEVICE; DOWN REGULATION; ENZYME REGULATION; HEART DILATATION; HEART GRAFT; HEART LEFT VENTRICLE ENDDIASTOLIC VOLUME; HEART TRANSPLANTATION; HUMAN; LUNG ARTERY PRESSURE; LUNG CAPILLARY PRESSURE; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; RADIOIMMUNOASSAY; STEADY STATE; TIME;

EID: 0034852267     PISSN: 10532498     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1053-2498(01)00289-3     Document Type: Article

References (22)

1. Stretch-induced atrial natriuretic factor release utilizes a rapidly depleting pool of newly synthesized hormone
2. Atrial stretch induces rapid increase in brain natriuretic peptide but not in atrial natriuretic peptide gene expression in vitro
3. Mechanical strain activates BNP gene transcription through a p38/NF-kappaB-dependent mechanism
4. Integrin dependence of brain natriuretic peptide gene promoter activation by mechanical strain
5. Mechanical circulatory support for advanced heart failure - Effect of patient selection on outcome
6. Mechanical circulatory support: The Bad Oeynhausen experience
7. Weaning from mechanical cardiac support in patients with idiopathic dilated cardiomyopathy
8. Myocyte recovery after mechanical circulatory support in humans with end-stage heart failure
9. Myocardial gene expression of regulators of myocyte apoptosis and myocyte calcium homeostasis during hemodynamic unloading by ventricular assist devices in patients with end-stage heart failure
10. Decreased expression of tumor necrosis factor-alpha in failing human myocardium after mechanical circulatory support: A potential mechanism for cardiac recovery
11. Analytical performance and clinical usefulness of a commercially available IRMA kit for measuring atrial natriuretic peptide in patients with heart failure
12. Localization and mechanism of secretion of B-type natriuretic peptide in comparison with those of A-type natriuretic peptide in normal subjects and patients with heart failure
13. Brain natriuretic peptide as a novel cardiac hormone in humans. Evidence for an exquisite dual natriuretic peptide system, atrial natriuretic peptide and brain natriuretic peptide
14. Superiority of brain natriuretic peptide as a hormonal marker of ventricular systolic and diastolic dysfunction and ventricular hypertrophy
15. Plasma brain natriuretic peptide level as a biochemical marker of morbidity and mortality in patients with asymptomatic or minimally symptomatic left ventricular dysfunction. Comparison with plasma angiotensin II and endothelin-1
16. Altered levels of mRNA of apoptosis-mediating genes after mid-term mechanical ventricular support in dilative cardiomyopathy - First results of the Halle Assist Induced Recovery Study (HAIR)
17. Atrial stretch, not pressure, is the principal determinant controlling the acute release of atrial natriuretic factor
18. Expression of atrial natriuretic factor in the human ventricle is independent of chamber dilation
19. Natriuretic peptide system in human heart failure
20. Different secretion patterns of atrial natriuretic peptide and brain natriuretic peptide in patients with congestive heart failure
21. Brain and atrial natriuretic peptides in patients with ischemic heart disease with and without heart failure
22. Mechanisms of mechanical load-induced atrial natriuretic peptide secretion: Role of endothelin, nitric oxide, and angiotensin II