Does epinephrine explain hyperlactatemia in cardiogenic shock? [1]

Critical Care Medicine

Volumn 29, Issue 9, 2001, Pages 1848-

  Luchette, F A ^a   James, J H ^a   Fischer, J E ^a  

^a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATASE; ADRENALIN; BETA ADRENERGIC RECEPTOR; CATECHOLAMINE; GLUCOSE; GLYCOGEN; INSULIN; LACTIC ACID; NORADRENALIN;

ADRENALIN BLOOD LEVEL; CARDIOGENIC SHOCK; ENZYME ACTIVITY; GLUCOSE INFUSION; GLYCOGEN SYNTHESIS; GLYCOLYSIS; HUMAN; HYPERGLYCEMIA; HYPERLACTATEMIA; INSULIN RESISTANCE; LACTATE BLOOD LEVEL; LACTIC ACIDOSIS; LETTER; MEAN ARTERIAL PRESSURE; OPEN HEART SURGERY; PRIORITY JOURNAL;

EID: 0034846818     PISSN: 00903493     EISSN: None     Source Type: Journal    
DOI: 10.1097/00003246-200109000-00038     Document Type: Letter

References (5)

1. Effects of cardiogenic shock on lactate and glucose metabolism after heart surgery
2. Lactic acidosis and insulin resistance associated with epinephrine administration in a patient with non-insulin-dependent diabetes mellitus
3. Beta 2-adrenoceptors mediate the stimulating effect of adrenaline on active electrogenic Na-K-transport in rat soleus muscle
4. +-ATPase activity in skeletal muscle by epinephrine or amylin
5. Lactate is an unreliable indicator of tissue hypoxia in injury or sepsis