An alopecic phenotype of canine pemphigus vulgaris? [6]

British Journal of Dermatology

Volumn 145, Issue 1, 2001, Pages 176-178

  Olivry, T ^a   Jackson, H A ^a  

^a NORTH CAROLINA STATE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMOXICILLIN PLUS CLAVULANIC ACID; DESMOGLEIN; IMMUNOGLOBULIN A; IMMUNOGLOBULIN G ANTIBODY; IMMUNOGLOBULIN M; PREDNISONE; PROTEIN SUBUNIT;

ACANTHOLYSIS; ALOPECIA; ANIMAL MODEL; ANIMAL TISSUE; ANTIGEN BINDING; CLINICAL FEATURE; CONTROLLED STUDY; DISEASE COURSE; DOG; ENZYME LINKED IMMUNOSORBENT ASSAY; FEMALE; GENE EXPRESSION; HAIR FOLLICLE; HAIR LOSS; HISTOPATHOLOGY; IMMUNOFLUORESCENCE TEST; IMMUNOGLOBULIN DEPOSITION; KERATINOCYTE; LETTER; NONHUMAN; PEMPHIGUS VULGARIS; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN EXPRESSION; SKIN BIOPSY; TREATMENT OUTCOME;

ALOPECIA; ANIMALS; AUTOANTIBODIES; DOG DISEASES; DOGS; FEMALE; PEMPHIGUS; PHENOTYPE;

EID: 0034825831     PISSN: 00070963     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1365-2133.2001.04311.x     Document Type: Letter

References (8)

1. The clinical phenotype of pemphigus is defined by the anti-desmoglein autoantibody profile
2. Compositionally different desmosomes in the various compartments of the human hair follicle
3. Desmoglein 3 anchors telogen hair in the follicle
4. Targeted disruption of the pemphigus vulgaris antigen (desmoglein 3) gene in mice causes loss of keratinocyte cell adhesion with a phenotype similar to pemphigus vulgaris
5. Vesicle formation and follicular root sheath separation in mice homozygous for deleterious alleles at the balding (bal) locus
6. Use of autoantigen-knockout mice in developing an active autoimmune disease model for pemphigus
7. Anti-plakin and desmoglein autoantibodies in a dog with pemphigus vulgaris
8. Normal anagen effluvium: A sign of pemphigus vulgaris