IL-9 stimulates release of chemotactic factors from human bronchial epithelial cells

American Journal of Respiratory Cell and Molecular Biology

Volumn 25, Issue 3, 2001, Pages 347-352

  Little, F F ^a   Cruikshank, W W ^a   Center, D M ^a  

^a BOSTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ASTHMA; CELL LINE; CHEMOATTRACTANT; CHEMOTACTIC FACTOR; EPITHELIUM CELL; FLUORESCENCE ACTIVATED CELL SORTING; HELPER CELL; HUMAN CELL; IMMUNE RESPONSE; INTERLEUKIN 9; MESSENGER RNA; RANTES; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SIGNAL TRANSDUCTION; T LYMPHOCYTE;

EID: 0034813106     PISSN: 10441549     EISSN: None     Source Type: Journal    
DOI: 10.1165/ajrcmb.25.3.4349     Document Type: Article

References (29)

1. Interleukin-9: A candidate gene for asthma
2. Role of IL-9 in the pathophysiology of allergic diseases
3. Genetic susceptibility to asthma: Bronchial hyperresponsiveness coinherited with a major gene for atopy
4. Allelic association of gene markers on chromosomes 5q and 11q with atopy and bronchial hyperresponsiveness
5. IL-9 pathway in asthma: New therapeutic targets for allergic inflammatory disorders
6. Interleukin-9 promotes allergen-induced eosinophilic inflammation and airway hyperresponsiveness in transgenic mice
7. IL-9 induces chemokine expression in lung epithelial cells and baseline airway eosinophilla in transgenic mice
8. Human P40/IL-9: Expression in activated CD4+ T cells, genomic organization, and comparison with the mouse gene
9. Allergen-induced IL-9 directly stimulates mucin transcription in respiratory epithelial cells
10. IL-9 and its receptor in allergic and nonallergic disease: Increased expression in asthma
11. Heteromerization of the γc chain with the interleukin-9 receptor α subunit leads to STAT activation and prevention of apoptosis
12. Interleukin-16: Implications for CD4 functions and HIV-1 progression
13. Structure and function of interleukin-16
14. Interleukin-16
15. Activation of dual T cell signaling pathways by the chemokine RANTES
16. Characterization of human bronchial epithelial cells immortalized by the E6 and E7 genes of human papillomavirus type 16
17. Transformation of human bronchial epithelial cells by infection with SV40 of adenovirus-12 SV40 hybrid virus, or transfection via strontium phosphate coprecipitation with a plasmid containing the SV40 early region genes
18. Reciprocal desensitization of CCR5 and CD4 is mediated by IL-16 and macrophage-inflammatory protein 1β, respectively
19. lck enzymatic activity
20. + T lymphocytes and monocytes
21. Early identification of interleukin-16 (lymphocyte chemoattractant factor) and macrophage inflammatory protein 1-α (MIP1-α) in bronchoalveolar lavage fluid of antigen-challenged asthmatics
22. Increased expression of interleukin-16 in bronchial mucosa of subjects with atopic asthma
23. Similar RANTES levels in healthy and asthmatic airways by immunoassay and in situ hybridization
24. Identification of IL-16 as the lymphocyte chemotactic activity in the bronchoalveolar lavage fluid of histamine-challenged asthmatic patients
25. Role of B7-CD28/CTLA-4 costimulation and NF-kappaB in allergen-induced T cell chemotaxis by IL-16 and RANTES
26. Interleukin-9 upregulates mucus expression in the airways
27. Interleukin (IL)-4/IL-9 and exogenous IL-16 induce IL-16 production by BEAS-2B cells, a bronchial epithelial cell line
28. Interleukin-9 receptor expression in asthmatic airways in vivo
29. IL-16 inhibits IL-5 production by antigen-stimulated T cells in atopic subjects