Biochemical basis for the hypercoagulable state seen in Cushing syndrome

Archives of Surgery

Volumn 136, Issue 9, 2001, Pages 1003-1007

  Jacoby, Robert C ^a   Owings, John T ^a   Ortega, Theresa ^b   Gosselin, Robert ^a   Feldman, Edward C ^b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BLOOD CLOTTING FACTOR 10; BLOOD CLOTTING FACTOR 12; BLOOD CLOTTING FACTOR 5; BLOOD CLOTTING FACTOR 7; BLOOD CLOTTING FACTOR 9; FIBRINOGEN; PLASMINOGEN; PROTHROMBIN; THROMBIN ANTITHROMBIN COMPLEX;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BLOOD LEVEL; CONTROLLED STUDY; CUSHING SYNDROME; DISEASE ASSOCIATION; DISEASE COURSE; DOG; HYPERCOAGULABILITY; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; THROMBOSIS;

EID: 0034779086     PISSN: 00040010     EISSN: None     Source Type: Journal    
DOI: 10.1001/archsurg.136.9.1003     Document Type: Article

References (15)

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5. Further studies on the hypercoagulable state of patients with Cushing's syndrome
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8. Complications following adrenal surgery
9. Complications and concurrent disease associated with canine hyperadrenocorticism
10. The scientific basis for selection of measures of thrombogenicity
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12. The fibrinolytic potential in patients with Cushing's disease: A clue to their hypercoagulable state
13. Venous thrombosis and tissue plasminogen activator release deficiency: A family study
14. Elevated plasminogen activator inhibitor PAI, a cause of thrombophilia? A study in 203 patients with familial or sporadic venous thrombophilia
15. Effect of critical injury on plasma antithrombin activity: Low antithrombin levels are associated with thromboembolic complications