Activation of inositol 1,4,5-trisphosphate receptor is essential for the opening of mouse TRP5 channels

Molecular Pharmacology

Volumn 60, Issue 5, 2001, Pages 989-998

  Kanki, Hideaki ^a   Kinoshita, Mariko ^a   Akaike, Akinori ^a   Satoh, Masamichi ^a   Mori, Yasuo ^b   Kaneko, Shuji ^a  

^a KYOTO UNIVERSITY   (Japan)

^b National Institutes of Natural Sciences   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

1 [[6 (3 METHOXYESTRA 1,3,5(10) TRIEN 17BETA YL)AMINO]HEXYL] 1H PYRROLE 2,5 DIONE; 1 [[6 (3 METHOXYESTRA 1,3,5(10) TRIEN 17BETA YL)AMINO]HEXYL] 2,5 PYRROLIDINEDIONE; ACETYLCHOLINE; ADENOPHOSTIN A; ANTISENSE OLIGODEOXYNUCLEOTIDE; CALCIUM CHANNEL; CALCIUM ION; CHLORIDE CHANNEL; ENZYME INHIBITOR; GUANINE NUCLEOTIDE BINDING PROTEIN; GUANOSINE 5' O (3 THIOTRIPHOSPHATE); INOSITOL 1,4,5 TRISPHOSPHATE RECEPTOR; MUSCARINIC M1 RECEPTOR; PROTEIN TRP5; RECEPTOR PROTEIN; RNA; THAPSIGARGIN; TRANSIENT RECEPTOR POTENTIAL PROTEIN; UNCLASSIFIED DRUG; XESTOSPONGIN C;

ANIMAL CELL; ARTICLE; CHLORIDE CURRENT; CONTROLLED STUDY; DRUG BINDING SITE; DRUG RECEPTOR BINDING; FEMALE; MOUSE; NONHUMAN; OOCYTE; PHOSPHOINOSITIDE METABOLISM; PRIORITY JOURNAL; XENOPUS;

EID: 0034752902     PISSN: 0026895X     EISSN: None     Source Type: Journal    
DOI: 10.1124/mol.60.5.989     Document Type: Article

References (40)

1. A transient calcium-dependent chloride current in the immature Xenopus oocyte
2. q/11, and phospholipase C-β1
3. Molecular cloning and immunolocalization of a novel vertebrate trp homologue from Xenopus
4. 2+ entry
5. 2+ inflow in Xenopus laevis oocytes
6. CRAC)
7. A long-lasting calcium-activated nonselective cationic current is generated by synaptic stimulation or exogenous activation of group I metabotropic glutamate receptors in CA1 pyramidal neurons
8. 2+ stores in the Xenopus oocyte
9. 2+ pumps
10. 2+ influx: How many mechanisms for how many channels
11. Molecular distinction between muscarinic acetylcholine receptor subtypes
12. Xestospongins: Potent membrane permeable blockers of the inositol 1,4,5-trisphosphate receptor
13. From worm to man: Three subfamilies of TRP channels
14. - currents in Xenopus laevis oocytes
15. Direct activation of human TRPC6 and TRPC3 channels by diacylglycerol
16. Metabotropic responses to acetylcholine and serotonin of Xenopus oocytes injected with rat brain mRNA are transduced by different G protein subtypes
17. 2+ in Xenopus oocytes expressing rat TRP4
18. 2+) channels
19. 3 receptor: Structure, function, and localization in oocytes and eggs
20. + channel determined by construction of multimeric cDNAs
21. 2+ channels
22. 3 receptor subtypes
23. Inositol phosphate formation and chloride current responses induced by acetylcholine and serotonin through GTP-binding proteins in Xenopus oocyte after injection of rat brain messenger RNA
24. 11 subunits
25. Molecular and functional characterization of a novel mouse transient receptor potential protein homologue TRP7
26. 2+ channel from mouse brain
27. o-type protein of Xenopus laevis oocytes
28. Molecular cloning and sequence determination of four different cDNA species coding for alpha-subunits of G proteins from Xenopus laevis oocytes
29. A novel capacitative calcium entry channel expressed in excitable cells
30. Receptor-mediated regulation of the nonselective cation channels TRPC4 and TRPC5
31. q mediate the response to trypsin in Xenopus oocytes
32. 11
33. 2+ release in Xenopus oocytes
34. TRPC1 and TRPC5 form a novel cation channel in mammalian brain
35. Adenophostins, newly discovered metabolites of Penicillium brevicompactum, act as potent agonists of the inositol 1,4,5-trisphosphate receptor
36. 3 receptors on the carboxyl termini of Trp channels
37. The aminosteroid U-73122 inhibits muscarinic receptor sequestration and phosphoinositide hydrolysis in SK-N-SH neuroblastoma cells
38. CaT1 manifests the pore properties of the calcium-release-activated calcium channel
39. Mutational analysis of the ligand binding site of the inositol 1,4,5-trisphosphate receptor
40. Activation of Trp3 by inositol 1,4,5-trisphosphate receptors through displacement of inhibitory calmodulin from a common binding domain