Increased neuronal excitability after long-term O2 deprivation is mediated mainly by sodium channels

Molecular Brain Research

Volumn 76, Issue 2, 2000, Pages 211-219

  Xia, Ying ^a   Fung, Man Lung ^a,c   O'Reilly, John P ^b   Haddad, Gabriel G ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b YALE UNIVERSITY   (United States)

^c UNIVERSITY OF HONG KONG   (Hong Kong)

Author keywords

Brain; Electrophysiology; Excitability; Hypoxia; Sodium channel; Tetrodotoxin

Indexed keywords

MESSENGER RNA; SAXITOXIN; SODIUM CHANNEL; TETRODOTOXIN;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; AUTORADIOGRAPHY; BRAIN CORTEX; BRAIN SLICE; CONTROLLED STUDY; ELECTROPHYSIOLOGY; HIPPOCAMPUS; HYPOXIA; NERVE EXCITABILITY; NONHUMAN; POSTNATAL DEVELOPMENT; PRIORITY JOURNAL; RAT; RNA ANALYSIS;

ANIMALS; ANIMALS, NEWBORN; ANOXIA; BRAIN; CELL MEMBRANE; CEREBRAL CORTEX; HIPPOCAMPUS; NEOCORTEX; NEURONS; RATS; RATS, SPRAGUE-DAWLEY; SAXITOXIN; SODIUM CHANNELS; TETRODOTOXIN; TRANSCRIPTION, GENETIC;

EID: 0034728575     PISSN: 0169328X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0169-328X(99)00338-1     Document Type: Article

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