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Volumn 87, Issue 11, 2000, Pages
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Mice with disrupted BK channel beta1 subunit gene feature abnormal Ca(2+) spark/STOC coupling and elevated blood pressure.
a
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Author keywords
[No Author keywords available]
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Indexed keywords
CALCIUM ACTIVATED POTASSIUM CHANNEL;
LARGE CONDUCTANCE CALCIUM ACTIVATED POTASSIUM CHANNEL;
MESSENGER RNA;
POTASSIUM;
POTASSIUM CHANNEL;
POTASSIUM CHLORIDE;
PROTEIN SUBUNIT;
VASODILATOR AGENT;
ANIMAL;
ARTICLE;
BRAIN ARTERY;
C57BL MOUSE;
CALCIUM SIGNALING;
CELL CULTURE;
CELL MEMBRANE POTENTIAL;
DNA SEQUENCE;
DRUG EFFECT;
FEMALE;
GENE TARGETING;
GENETICS;
HOMOZYGOTE;
HYPERTENSION;
IN VITRO STUDY;
MALE;
METABOLISM;
MOUSE;
MOUSE MUTANT;
SIGNAL TRANSDUCTION;
VASCULAR SMOOTH MUSCLE;
VASOCONSTRICTION;
ANIMALS;
CALCIUM SIGNALING;
CELLS, CULTURED;
CEREBRAL ARTERIES;
FEMALE;
GENE TARGETING;
HOMOZYGOTE;
HYPERTENSION;
LARGE-CONDUCTANCE CALCIUM-ACTIVATED POTASSIUM CHANNELS;
MALE;
MEMBRANE POTENTIALS;
MICE;
MICE, INBRED C57BL;
MICE, KNOCKOUT;
MUSCLE, SMOOTH, VASCULAR;
POTASSIUM;
POTASSIUM CHANNELS;
POTASSIUM CHANNELS, CALCIUM-ACTIVATED;
POTASSIUM CHLORIDE;
PROTEIN SUBUNITS;
RNA, MESSENGER;
SEQUENCE ANALYSIS, DNA;
SIGNAL TRANSDUCTION;
VASOCONSTRICTION;
VASODILATOR AGENTS;
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EID: 0034711349
PISSN: None
EISSN: 15244571
Source Type: Journal
DOI: None Document Type: Article |
Times cited : (315)
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References (0)
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