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Volumn 38, Issue 4, 2000, Pages 384-391
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Do synaptic rearrangements underlie compensatory reflex enhancement in spinal motoneurons after partial cell loss?
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Author keywords
Adult mammals; Axonal lesion; Retrograde cell death; Synaptic plasticity
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Indexed keywords
HORSERADISH PEROXIDASE;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
AVULSION INJURY;
CAT;
CELL DEATH;
CELL LOSS;
CONTROLLED STUDY;
ELECTRON MICROSCOPY;
MITOCHONDRION;
MONOSYNAPTIC REFLEX;
MOTONEURON NUCLEUS;
NERVE CELL PLASTICITY;
NONHUMAN;
PRIORITY JOURNAL;
SPINAL CORD MOTONEURON;
SPINAL CORD VENTRAL HORN;
SPINAL GANGLION;
SYNAPTIC POTENTIAL;
VENTRAL ROOT;
ADAPTATION, PHYSIOLOGICAL;
ANIMALS;
CATS;
CELL DEATH;
CHOLERA TOXIN;
GANGLIA, SPINAL;
HORSERADISH PEROXIDASE;
LUMBOSACRAL REGION;
MICROSCOPY, ELECTRON;
MOTOR NEURONS;
NEURONAL PLASTICITY;
PRESYNAPTIC TERMINALS;
REFLEX;
SPINAL CORD;
SYNAPSES;
WHEAT GERM AGGLUTININ-HORSERADISH PEROXIDASE CONJUGATE;
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EID: 0034672815
PISSN: 08874476
EISSN: None
Source Type: Journal
DOI: 10.1002/1098-2396(20001215)38:4<384::AID-SYN3>3.0.CO;2-X Document Type: Article |
Times cited : (10)
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References (37)
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