Acrolein, a product of lipid peroxidation, inhibits glucose and glutamate uptake in primary neuronal cultures

Free Radical Biology and Medicine

Volumn 29, Issue 8, 2000, Pages 714-720

  Lovell, Mark A ^a,b   Xie, Chengsong ^a   Markesbery, William R ^a,b  

^a UNIVERSITY OF KENTUCKY   (United States)

^b UNIVERSITY OF KENTUCKY   (United States)

Author keywords

Alzheimer's disease; Free radicals; Lipid peroxidation; Neuronal cultures; Neurotoxin

Indexed keywords

ACROLEIN; GLUCOSE; GLUTAMIC ACID;

ALZHEIMER DISEASE; AMYGDALOID NUCLEUS; ANIMAL CELL; ARTICLE; ASTROCYTE; BRAIN CELL; CONTROLLED STUDY; GLUCOSE TRANSPORT; LIPID PEROXIDATION; NERVE CELL DEGENERATION; NEUROTOXICITY; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; SUBICULUM;

ACROLEIN; ALZHEIMER DISEASE; ANIMAL; BIOLOGICAL TRANSPORT, ACTIVE; CELLS, CULTURED; CEREBRAL CORTEX; GLUCOSE; GLUTAMIC ACID; HUMAN; LIPID PEROXIDATION; NERVE DEGENERATION; NEURONS; OXIDATIVE STRESS; RATS; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0034667741     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0891-5849(00)00346-4     Document Type: Article

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