A case-control study confirms that microsatellite assay can identify patients at risk of developing oral squamous cell carcinoma within a field of cancerization

Cancer Research

Volumn 60, Issue 14, 2000, Pages 3893-3898

  Partridge, Maxine ^a   Pateromichelakis, Stelios ^a   Phillips, Elaine ^a   Emilion, Gracy G ^a   A’Hern, Roger P ^b   Langdon, John D ^a  

^a KING S COLLEGE HOSPITAL   (United Kingdom)

^b INSTITUTE OF CANCER RESEARCH   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ADULT; AGED; ARTICLE; CANCER RISK; CARCINOGENESIS; CASE CONTROL STUDY; CLINICAL ARTICLE; CONTROLLED STUDY; DYSPLASIA; FEMALE; HUMAN; MALE; MICROSATELLITE INSTABILITY; MOUTH CARCINOMA; PRIORITY JOURNAL;

ADULT; AGED; ALLELES; CARCINOMA, SQUAMOUS CELL; CASE-CONTROL STUDIES; FEMALE; HUMANS; MALE; MICROSATELLITE REPEATS; MIDDLE AGED; MOUTH MUCOSA; MOUTH NEOPLASMS; PRECANCEROUS CONDITIONS; PROGNOSIS; RISK FACTORS;

EID: 0034660881     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (31)

1. Oral leukoplakia: Rationale for diagnosis and prognosis of its clinical subtypes or "phases"
2. Definition of leukoplakia and related lesions: An aid to studies on oral precancer
3. Leukoplakia revisited: A clinicopathologic study of 3256 oral leukoplakias
4. Premalignant lesions of oral epithelium
5. Oral leukoplakia and malignant transformation: A follow-up study of 257 patients
6. Oral epithelial dysplasia and the development of invasive squamous cell carcinoma
7. Erythroplakia of the oral cavity
8. Klinik oraler leukoplakien und prakanzerosen retrospektive studie an 200 patienten
9. Basic histopathological features of oral premalignant lesions
10. Observer variability in the histologic assessment of oral premalignant lesions
11. A genetic model for colorectal tumorigenesis
12. LOH at 3p correlates with a poor survival in oral squamous cell carcinoma
13. Frequent microsatellite alterations at chromosomes 9p21 and 3p14 in oral premalignant lesions and their value in cancer risk assessment
14. Loss of heterozygosity on the short arm of chromosomes 3 and 9 in oral cancel
15. Allelic imbalance on chromosome 3p in oral dysplastic lesions: An early event in oral carcinogenesis
16. Deletion mapping defines three discrete areas of allelic imbalance on chromosome arm 8p in oral and oropharyngeal squamous cell carcinomas
17. FHIT gene alterations in head and neck squamous cell carcinomas
18. PRAD-1 oncogene amplification in primary head and neck squamous cell carcinoma
19. Genetic progression model for head and neck cancer: Implications for field cancerization
20. Allelic imbalance at chromosomal loci implicated in the pathogenesis of oral precancer; cumulative loss and its relationship with progression to cancer
21. Sequential loss of heterozygosity at microsatellite motifs in preinvasive and invasive head and neck squamous carcinoma
22. Frequent gene deletions in potentially malignant oral lesions
23. Patient-specific mutation databases for oral cancer
24. INK4ain oral premalignant lesions
25. Molecular damage in the bronchial epithelium of current and former smokers
26. Pussycats and baby tigers: Non-invasive (pTa) and minimally invasive (pTI) bladder carcinomas are not the same
27. Detection of chromosome instability of tissue fields at risk: In situ hybridization
28. Allele-specific chromosome 3p deletions occur at an early stage in the pathogenesis of lung carcinoma
29. Widely dispersed p53 mutations in respiratory epithelium. A novel mechanism for field carcinogenesis
30. Phenotype and genotype of advanced premalignant head and neck lesions after chemopreventive therapy
31. Detection of minimal residual cancer to investigate why tumors recur despite seemingly adequate treatment