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Volumn 17, Issue 6, 2000, Pages 1179-1185
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Inhibition of oncogene STAT3 phosphorylation by a prolactin antagonist, hPRL-G129R, in T-47D human breast cancer cells.
a
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Author keywords
[No Author keywords available]
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Indexed keywords
ANTINEOPLASTIC HORMONE AGONISTS AND ANTAGONISTS;
DNA BINDING PROTEIN;
GROWTH HORMONE RECEPTOR;
HORMONE ANTAGONIST;
HUMAN GROWTH HORMONE;
HYBRID PROTEIN;
MILK PROTEIN;
PROLACTIN;
PROLACTIN RECEPTOR;
PROLACTIN, ARG(129);
PROLACTIN, ARG(129)-;
STAT3 PROTEIN;
STAT3 PROTEIN, HUMAN;
STAT3 PROTEIN, MOUSE;
STAT5 PROTEIN;
TRANSACTIVATOR PROTEIN;
TUMOR PROTEIN;
AMINO ACID SUBSTITUTION;
ANIMAL;
ARTICLE;
BREAST TUMOR;
CELL CULTURE;
COMPARATIVE STUDY;
DOSE RESPONSE;
DRUG ANTAGONISM;
DRUG EFFECT;
FEMALE;
GENETICS;
HUMAN;
L CELL;
METABOLISM;
MOUSE;
ONCOGENE;
PATHOLOGY;
PHOSPHORYLATION;
PROTEIN PROCESSING;
AMINO ACID SUBSTITUTION;
ANIMALS;
ANTINEOPLASTIC AGENTS, HORMONAL;
BREAST NEOPLASMS;
DNA-BINDING PROTEINS;
DOSE-RESPONSE RELATIONSHIP, DRUG;
FEMALE;
HORMONE ANTAGONISTS;
HUMAN GROWTH HORMONE;
HUMANS;
L CELLS (CELL LINE);
MICE;
MILK PROTEINS;
NEOPLASM PROTEINS;
ONCOGENES;
PHOSPHORYLATION;
PROLACTIN;
PROTEIN PROCESSING, POST-TRANSLATIONAL;
RECEPTORS, PROLACTIN;
RECEPTORS, SOMATOTROPIN;
RECOMBINANT FUSION PROTEINS;
STAT3 TRANSCRIPTION FACTOR;
STAT5 TRANSCRIPTION FACTOR;
TRANS-ACTIVATORS;
TUMOR CELLS, CULTURED;
MLCS;
MLOWN;
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EID: 0034547753
PISSN: 10196439
EISSN: None
Source Type: Journal
DOI: 10.3892/ijo.17.6.1179 Document Type: Article |
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Times cited : (42)
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References (0)
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