Relative sparing of the parietal cortex in cerebellar ataxia documented by positron emission tomography

Clinical Neurology and Neurosurgery

Volumn 102, Issue 4, 2000, Pages 210-214

  Rudolf, Jobst ^a,b   Grond, Martin ^a,b   Hilker, Ruediger ^a   Ghaemi, Mehran ^a   Jacobs, Andreas ^b   Heiss, Wolf Dieter ^a,b  

^a UNIVERSITY HOSPITAL OF COLOGNE   (Germany)

^b MAX PLANCK INSTITUTE FOR NEUROLOGICAL RESEARCH   (Germany)

Author keywords

2 18F fluoro 2 deoxy D glucose; Cerebral glucose metabolism; Hereditary cerebellar ataxia; Positron emission tomography

Indexed keywords

FLUORODEOXYGLUCOSE;

ADULT; AGED; ARTICLE; BRAIN REGION; CEREBELLAR ATAXIA; CLINICAL ARTICLE; COMPUTER ASSISTED TOMOGRAPHY; CONTROLLED STUDY; FEMALE; GLUCOSE METABOLISM; HEMISPHERE; HEREDITARY ATAXIA; HUMAN; MALE; PARIETAL LOBE; POSITRON EMISSION TOMOGRAPHY; SYMPTOM;

ADULT; AGED; CEREBELLAR ATAXIA; FEMALE; FLUORODEOXYGLUCOSE F18; GLUCOSE; HUMANS; MALE; MIDDLE AGED; PARIETAL LOBE; RADIOPHARMACEUTICALS; TOMOGRAPHY, EMISSION-COMPUTED;

EID: 0034538962     PISSN: 03038467     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0303-8467(00)00113-X     Document Type: Article

References (13)

1. Clinical features and classification of inherited ataxias
2. Genes involved in hereditary ataxias
3. Cinical and imaging correlations in inherited ataxias
4. Patterns of cerebral glucose metabolism detected with positron emission tomography differ in multiple system atrophy and olivopontocerebellar atrophy
5. The (18F)fluorodeoxyglucose method for the measurement of local cerebral glucose utilization in man
6. Regional kinetic constants and cerebral metabolic rate for glucose in normal human volunteers determined by dynamic positron emission tomography of (18F)-2-fluoro-2-deoxy-d-glucose
7. Computer assisted mapping in quantitative analysis of cerebral positron emission tomograms
8. Speech disorders in olivopontocerebellar atrophy correlate with positron emission tomography findings
9. Metabolic and cognitive changes in hereditary ataxia
10. Cerebellar and brainstem hypometabolism in olivopontocerebellar atrophy detected with positron emission tomography
11. Cerebral glucose hypermetabolism in Friedreich's ataxia detected with positron emission tomography
12. Motor dysfunction in olivopontocerebellar atrophy is related to cerebral metabolic rate studied with positron emission tomography
13. Striatal 18F-Dopa uptake and brain glucose metabolism by PET in patients with progressive ataxia