Decreased nitric oxide availability contributes to acute cerebral ischemia after subarachnoid hemorrhage

Neurosurgery

Volumn 47, Issue 1, 2000, Pages 208-215

  Schwartz, A Y ^a   Sehba, F A ^a   Bederson, J B ^a   Dacey, R G ^a   Dietrich, H ^a  

^a MOUNT SINAI SCHOOL OF MEDICINE   (United States)

Author keywords

Cerebral ischemia; NG nitro L arginine methyl ester; Nitric oxide; Nitric oxide synthase inhibitor; Rat; Subarachnoid hemorrhage; Vasoconstriction

Indexed keywords

N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; NITRIC OXIDE SYNTHASE INHIBITOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTERIAL PRESSURE; ARTICLE; BRAIN BLOOD FLOW; BRAIN ISCHEMIA; CONTROLLED STUDY; INTRACRANIAL PRESSURE; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; SUBARACHNOID HEMORRHAGE; TIME; VASOCONSTRICTION; VASODILATATION;

ACUTE DISEASE; ANIMALS; BLOOD PRESSURE; BRAIN ISCHEMIA; CEREBROVASCULAR CIRCULATION; ENZYME INHIBITORS; MALE; NG-NITROARGININE METHYL ESTER; NITRIC OXIDE; RATS; RATS, SPRAGUE-DAWLEY; SUBARACHNOID HEMORRHAGE;

EID: 0034519628     PISSN: 0148396X     EISSN: None     Source Type: Journal    
DOI: 10.1097/00006123-200007000-00042     Document Type: Article

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