Parovirus B19-associated mono- and oligoarticular arthritis may evolve into a chrome inflammatory arthropathy fulfilling criteria for rheumatoid arthritis or spondylarthropathy [1]

Clinical Rheumatology

Volumn 19, Issue 6, 2000, Pages 510-511

  Stahl, H D ^a   Pfeiffer, R ^a   Von Salis Soglio, G ^a   Emmrich, F ^a  

^a UNIVERSITY OF LEIPZIG   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

VIRUS DNA;

ADULT; ARTHRITIS; ARTHROPATHY; CLINICAL ARTICLE; DISEASE ACTIVITY; DISEASE COURSE; FEMALE; FOLLOW UP; HUMAN; IMMUNOPATHOLOGY; LETTER; MALE; PARVOVIRUS; PRIORITY JOURNAL; RHEUMATOID ARTHRITIS; SPONDYLOARTHROPATHY; SYNOVIUM;

ADULT; ARTHRITIS, INFECTIOUS; ARTHRITIS, RHEUMATOID; DISEASE PROGRESSION; DNA; FEMALE; HUMANS; JOINTS; MALE; PARVOVIRIDAE INFECTIONS; PARVOVIRUS B19, HUMAN; SPONDYLITIS; TREATMENT OUTCOME;

EID: 0034507543     PISSN: 07703198     EISSN: None     Source Type: Journal    
DOI: 10.1007/s100670070021     Document Type: Letter

References (7)

1. High incidence of parvovirus B19 DNA in synovial tissue of patients with undifferentiated mono- and oligoarthritis
2. Undifferentiated arthritis and spondylarthropathy as a major problem of diagnosis and classification
3. The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis
4. The European Spondylarthropathy Study Group preliminary criteria for the classification of spondylarthropathy
5. Human parvovirus B19 as a causative agent for rheumatoid arthritis
6. Persistence of parvovirus B19 DNA in synovial membranes of young patients with and without chronic arthropathy
7. Viral and bacterial infections interfere with peripheral tolerance induction and activate CD8+ T cells to cause immuopathology