Severity of hypertension in familial hyperaldosteronism type I: Relationship to gender and degree of biochemical disturbance

Journal of Clinical Endocrinology and Metabolism

Volumn 85, Issue 6, 2000, Pages 2160-2166

  Stowasser, M ^a   Bachmann, A W ^a   Huggard, P R ^a   Rossetti, T R ^a   Gordon, R D ^a  

^a UNIVERSITY OF QUEENSLAND   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

ADULT; ARTICLE; AUTOSOMAL DOMINANT INHERITANCE; BLOOD PRESSURE REGULATION; CATALYSIS; CIRCADIAN RHYTHM; CLINICAL ARTICLE; DISEASE SEVERITY; ENZYME ACTIVITY; FEMALE; GENETIC REGULATION; HUMAN; HYPERALDOSTERONISM; HYPERTENSION; MALE; MULTIGENE FAMILY; PRIORITY JOURNAL; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM; SEX DIFFERENCE;

ADOLESCENT; ADRENOCORTICOTROPIC HORMONE; ADULT; AGED; ALDOSTERONE; ALDOSTERONE SYNTHASE; CIRCADIAN RHYTHM; FEMALE; HUMANS; HYDROCORTISONE; HYPERALDOSTERONISM; HYPERTENSION; MALE; MIDDLE AGED; POSTURE; POTASSIUM; RENIN; SEX CHARACTERISTICS; SODIUM;

EID: 0034458423     PISSN: 0021972X     EISSN: None     Source Type: Journal    
DOI: 10.1210/jc.85.6.2160     Document Type: Article

References (32)

1. A chimaeric 11β-hydroxylase/aldosterone synthase gene causes glucocorticoid-remediable aldosteronism and human hypertension
2. In familial hyperaldosteronism type I, hybrid gene-induced aldosterone production dominates that induced by wild type genes
3. Anomalous postural aldosterone response in glucocorticoid-suppressible hyperaldosteronism
4. A new family with dexamethasone-suppressible hyperaldosteronism: Aldosterone unresponsiveness to angiotensin II
5. Evidence for an unidentified ACTH-induced steroid hormone causing hypertension
6. Familial, dexamethasone-suppressible, normokalemic hyperaldosteronism
7. Dexamethasone-suppressible hyperaldosteronism: Adrenal transitional cell hyperplasia?
8. Renin-aldosterone response to dexamethasone in glucocorticoid-suppressible hyperaldosteronism is altered by coexistent renal artery stenosis
9. Defective fasciculata zone function as the mechanism of glucocorticoid-remediable aldosteronism
10. Hybrid gene or hybrid steroids in the detection and screening for familial hyperaldosteronism type I
11. Glucocorticoid-remediable aldosteronism in a large kindred: Clinical spectrum and diagnosis using a characteristic biochemical phenotype
12. A PCR-based method of screening individuals of all ages, from neonates to the elderly, for familial hyperaldosteronism type I
13. Intracranial aneurysm and hemorrhagic stroke in glucocorticoid-remediable aldosteronism
14. Rapid diagnosis of glucocorticoid suppressible hyperaldosteronism in infants and adolescents
15. Glucocorticoid-suppressible hyperaldosteronism: Effects of crossover site and parental origin of chimaeric gene on phenotypic expression
16. Clinical, biochemical and genetic approaches to the detection of familial hyperaldosteronism type I
17. Variation of phenotype in patients with glucocorticoid-remediable aldosteronism
18. Familial forms broaden the horizons for primary aldosteronism
19. A new genetic test for familial hyperaldosteronism type I aids in the detection of curable hypertension
20. A PCR-based method of screening individuals of all ages, from neonates to the elderly, for familial hyperaldosteronism type I
21. Biochemical evidence of aldosterone overproduction and abnormal regulation in normotensive individuals with familial hyperaldosteronism type I
22. Report of the second task force on blood pressure control in children-1987
23. A radioimmunoassay for plasma aldosterone
24. Application of a radioimmunoassay for angiotensin I to the physiologic measurement of plasma renin activity in normal human subjects
25. Glucocorticoid-remediable aldosteronism (GRA): Diagnosis, variability of phenotype and regulation of potassium homeostasis
26. Plasma renin activity and plasma prorenin assays
27. The effect of chronic adrenergic receptor blockade on plasma renin activity in man
28. Converting-enzyme inhibitors in the treatment of hypertension
29. Effects of nifedipine treatment on the renin-angiotensin-aldosterone axis
30. Receptor binding and biological activity of 18-oxocortisol
31. Alteration by oral contraceptives of normal menstrual changes in plasma renin activity, concentration and substrate
32. Plasma renin concentration, activity, and substrate in hypertension induced by oral contraceptives