Platelets from a patient heterozygous for the defect of P2CYC receptors for ADP have a secretion defect despite normal thromboxane A2 production and normal granule stores: further evidence that some cases of platelet 'primary secretion defect' are heterozygous for a defect of P2CYC receptors.

Arteriosclerosis, thrombosis, and vascular biology

Volumn 20, Issue 11, 2000, Pages

  Cattaneo, M ^a   Lecchi, A ^a   Lombardi, R ^a   Gachet, C ^a   Zighetti, M L ^a  

^a UNIVERSITY OF MILAN   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE DIPHOSPHATE; ADENOSINE TRIPHOSPHATE; PURINE P2 RECEPTOR; THROMBOXANE A2;

ADOLESCENT; ADULT; ARTICLE; BIOSYNTHESIS; BLOOD; CASE REPORT; CELL GRANULE; FEMALE; GENETICS; HETEROZYGOTE DETECTION; HUMAN; MALE; METABOLISM; MIDDLE AGED; PATHOLOGY; SECRETION; THROMBOCYTE; THROMBOCYTE AGGREGATION; THROMBOCYTE DISORDER;

ADENOSINE DIPHOSPHATE; ADENOSINE TRIPHOSPHATE; ADOLESCENT; ADULT; BLOOD PLATELET DISORDERS; BLOOD PLATELETS; CYTOPLASMIC GRANULES; FEMALE; HETEROZYGOTE DETECTION; HUMANS; MALE; MIDDLE AGED; PLATELET AGGREGATION; RECEPTORS, PURINERGIC P2; THROMBOXANE A2;

EID: 0034323126     PISSN: None     EISSN: 15244636     Source Type: Journal    
DOI: None     Document Type: Article

References (0)