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Volumn 2, Issue 3, 2000, Pages 295-301

Aldosterone-related genetic effects in hypertension

Author keywords

[No Author keywords available]

Indexed keywords

ALDOSTERONE; ALDOSTERONE SYNTHASE; MINERALOCORTICOID; RENIN; SODIUM; SODIUM CHANNEL;

EID: 0034203243     PISSN: 15226417     EISSN: 15343111     Source Type: Journal    
DOI: 10.1007/s11906-000-0013-3     Document Type: Article
Times cited : (6)

References (53)
  • 1
    • 0025817650 scopus 로고
    • Blood pressure control: Special role of kidneys and body fluids
    • Guyton AC: Blood pressure control: special role of kidneys and body fluids. Science 1991, 252: 1813-1816.
    • (1991) Science , vol.252 , pp. 1813-1816
    • Guyton, A.C.1
  • 2
    • 0033560346 scopus 로고    scopus 로고
    • Genetic disorders of renal electrolyte transport
    • Scheinman SJ, Guay-Woodford L M, Thakker RV, et al.: Genetic disorders of renal electrolyte transport. N Engl J Med 1999, 340: 1177-1187. This recent review is authoratative and trenchant, and it provides an extensive listing of renal tubule transport disorders with relevant links to the On-Line Mendelian Inheritance in Man.
    • (1999) N Engl J Med , vol.340 , pp. 1177-1187
    • Scheinman, S.J.1    Guay-Woodford, L.M.2    Thakker, R.V.3
  • 3
    • 0027946089 scopus 로고
    • Liddle's syndrome: Heritable human hypertension caused by mutations in the b subunit of the epithelial sodium channel
    • Shimkets RA, Warnock DG, Bositis CM, et al.: Liddle's syndrome: heritable human hypertension caused by mutations in the b subunit of the epithelial sodium channel. Cell 1994, 79: 407-414.
    • (1994) Cell , vol.79 , pp. 407-414
    • Shimkets, R.A.1    Warnock, D.G.2    Bositis, C.M.3
  • 4
    • 0030928166 scopus 로고    scopus 로고
    • Genotype-phenotype analysis of a newly discovered family with Liddle's syndrome
    • Jeunemaitre X, Bassilana F, Persu A, et al.: Genotype-phenotype analysis of a newly discovered family with Liddle's syndrome. J Hypertens 1997, 15: 1091-1100.
    • (1997) J Hypertens , vol.15 , pp. 1091-1100
    • Jeunemaitre, X.1    Bassilana, F.2    Persu, A.3
  • 5
    • 0033105864 scopus 로고    scopus 로고
    • Defective regulation of the epithelial Na+ channel by NEDD4 in Liddle's syndrome
    • Abriel H, Loffing H, Rebhus JF, et al.: Defective regulation of the epithelial Na+ channel by NEDD4 in Liddle's syndrome. J Clin Invest 1999, 103: 667-673. Elegant, definitive studies of the binding interactions between NEDD4 and the ENaC subunits.
    • (1999) J Clin Invest , vol.103 , pp. 667-673
    • Abriel, H.1    Loffing, H.2    Rebhus, J.F.3
  • 6
    • 0029586683 scopus 로고
    • A de novo missense mutation of the b subunit of the epithelial sodium channel causes hypertension and Liddle syndrome, identifying a proline-rich segment critical for regulation of channel activity
    • Hansson JH, Schild L, Lu Y, et al.: A de novo missense mutation of the b subunit of the epithelial sodium channel causes hypertension and Liddle syndrome, identifying a proline-rich segment critical for regulation of channel activity. Proc Natl Acad Sci U S A 1995, 92: 11495-11499.
    • (1995) Proc Natl Acad Sci U S A , vol.92 , pp. 11495-11499
    • Hansson, J.H.1    Schild, L.2    Lu, Y.3
  • 7
    • 0029918734 scopus 로고    scopus 로고
    • Liddle disease caused by a missense mutation of b subunit of the epithelial sodium channel gene
    • Tamura H, Schild L, Enomoto N, et al.: Liddle disease caused by a missense mutation of b subunit of the epithelial sodium channel gene. J Clin Invest 1996, 97: 1780-1784.
    • (1996) J Clin Invest , vol.97 , pp. 1780-1784
    • Tamura, H.1    Schild, L.2    Enomoto, N.3
  • 8
    • 0001182641 scopus 로고
    • A familial renal disorder simulating primary aldosteronism but with negligible aldosterone secretion
    • Liddle GW, Bledsoe T, Coppage WS: A familial renal disorder simulating primary aldosteronism but with negligible aldosterone secretion. Trans Am Assoc Physicians 1963, 76: 199-213.
    • (1963) Trans Am Assoc Physicians , vol.76 , pp. 199-213
    • Liddle, G.W.1    Bledsoe, T.2    Coppage, W.S.3
  • 9
    • 0029049742 scopus 로고
    • Liddle's syndrome: An under-recognized entity; a report of four cases including the first report in black individuals
    • Gadallah MF, Abreo K, Work J: Liddle's syndrome: an under-recognized entity; a report of four cases including the first report in black individuals. Am J Kidney Dis 1995, 25: 924-927.
    • (1995) Am J Kidney Dis , vol.25 , pp. 924-927
    • Gadallah, M.F.1    Abreo, K.2    Work, J.3
  • 10
    • 0031927840 scopus 로고    scopus 로고
    • Genetic analysis of the b subunit of the epithelial Na+ channel in essential hypertension
    • Persu A, Barbry P, Bassilana F, et al.: Genetic analysis of the b subunit of the epithelial Na+ channel in essential hypertension. Hypertension 1998, 32: 129-137.
    • (1998) Hypertension , vol.32 , pp. 129-137
    • Persu, A.1    Barbry, P.2    Bassilana, F.3
  • 11
    • 0029046975 scopus 로고
    • A mutation in the epithelial sodium channel causing Liddle disease increases channel activity in the Xenopus laevis oocyte expression sytem
    • Schild L, Canessa CM, Shimkets RA, et al.: A mutation in the epithelial sodium channel causing Liddle disease increases channel activity in the Xenopus laevis oocyte expression sytem. Proc Natl Acad Sci U S A 1995, 92: 5699-5703.
    • (1995) Proc Natl Acad Sci U S A , vol.92 , pp. 5699-5703
    • Schild, L.1    Canessa, C.M.2    Shimkets, R.A.3
  • 12
    • 0030405694 scopus 로고    scopus 로고
    • A novel variant of the b-subunit of the amiloride-sensitive sodium channel in African Americans
    • Su YR, Rutkowski MP, Klanke CA, et al.: A novel variant of the b-subunit of the amiloride-sensitive sodium channel in African Americans. J Am Soc Nephrol 1996, 7: 2543-2549.
    • (1996) J Am Soc Nephrol , vol.7 , pp. 2543-2549
    • Su, Y.R.1    Rutkowski, M.P.2    Klanke, C.A.3
  • 13
    • 0028228345 scopus 로고
    • G-proteins modulate amiloride-sensitive sodium channels
    • Bubien JK, Jope RS, Warnock DG: G-proteins modulate amiloride-sensitive sodium channels. J Biol Chem 1994, 269: 17780-17783.
    • (1994) J Biol Chem , vol.269 , pp. 17780-17783
    • Bubien, J.K.1    Jope, R.S.2    Warnock, D.G.3
  • 14
    • 0030667948 scopus 로고    scopus 로고
    • Expression of the amiloride-sensitive sodum channel b subunit gene in human B lymphocytes
    • Oh Y, Warnock DG: Expression of the amiloride-sensitive sodum channel b subunit gene in human B lymphocytes. J Am Soc Nephrol 1997, 8: 126-129.
    • (1997) J Am Soc Nephrol , vol.8 , pp. 126-129
    • Oh, Y.1    Warnock, D.G.2
  • 15
    • 12644278872 scopus 로고    scopus 로고
    • Loss of protein kinase C inhibition in the β-T594M variant of the amiloride-sensitive Na+ channel
    • Cui Y, Su YR, Rutkowski M, et al.: Loss of protein kinase C inhibition in the β-T594M variant of the amiloride-sensitive Na+ channel. Proc Natl Acad Sci U S A 1997, 94: 9962-9966.
    • (1997) Proc Natl Acad Sci U S A , vol.94 , pp. 9962-9966
    • Cui, Y.1    Su, Y.R.2    Rutkowski, M.3
  • 16
    • 0030378049 scopus 로고    scopus 로고
    • Editorial: Polymorphism in the beta subunit and Na+ transport
    • Warnock DG: Editorial: polymorphism in the beta subunit and Na+ transport. J Am Soc Nephrol 1996, 7: 2490-2494.
    • (1996) J Am Soc Nephrol , vol.7 , pp. 2490-2494
    • Warnock, D.G.1
  • 17
    • 0032499488 scopus 로고    scopus 로고
    • Association of hypertension with T594M mutation in the b subunit of epithelial sodium channels in black people resident in London
    • Baker EH, Dong YB, Sagnella GA, et al.: Association of hypertension with T594M mutation in the b subunit of epithelial sodium channels in black people resident in London. Lancet 1998, 351: 1388-1392.
    • (1998) Lancet , vol.351 , pp. 1388-1392
    • Baker, E.H.1    Dong, Y.B.2    Sagnella, G.A.3
  • 18
    • 85058248886 scopus 로고    scopus 로고
    • T594M mutation in the ENaC beta subunit and low-renin hypertension in blacks: Author's reply
    • Baker EH, Dong YB, Sagnella GA, et al.: T594M mutation in the ENaC beta subunit and low-renin hypertension in blacks: author's reply. Am J Kidney Dis 1999, 34: 583-587. Lively discussion of the case-control studies that demonstrated the association of the b subunit M594T mutation and low-renin hypertension in persons of African ancestry.
    • (1999) Am J Kidney Dis , vol.34 , pp. 583-587
    • Baker, E.H.1    Dong, Y.B.2    Sagnella, G.A.3
  • 19
    • 0032865808 scopus 로고    scopus 로고
    • T594M mutation in the ENaC beta subunit and low-renin hypertension in blacks: Journal club
    • Editorial comment on Baker et al. 18
    • Warnock D: T594M mutation in the ENaC beta subunit and low-renin hypertension in blacks: journal club. Am J Kidney Dis 1999, 34: 579-587. Editorial comment on Baker et al. [18].
    • (1999) Am J Kidney Dis , vol.34 , pp. 579-587
    • Warnock, D.1
  • 20
    • 0032702619 scopus 로고    scopus 로고
    • Genetic variants in the epithelial sodium channel in relation to aldosterone and potassium excretion and risk for hypertension
    • Ambrosius WT, Bloem LJ, Zhou L, et al.: Genetic variants in the epithelial sodium channel in relation to aldosterone and potassium excretion and risk for hypertension. Hypertension 1999, 34: 631-637. Surperb study of the relations among plasma renin, aldosterone, and ENaC polymorphisms. Examines the urinary aldosterone-potassium excretory ratio as a surrogate marker for ENaC activation in humans.
    • (1999) Hypertension , vol.34 , pp. 631-637
    • Ambrosius, W.T.1    Bloem, L.J.2    Zhou, L.3
  • 21
    • 0000230888 scopus 로고    scopus 로고
    • The T594M mutation in the b subunit of amiloride-sensitive sodium channel is associated with increased levels of urinary calcium excretion
    • Blackwood A, Baker EH, Dong YB, et al.: The T594M mutation in the b subunit of amiloride-sensitive sodium channel is associated with increased levels of urinary calcium excretion. Hypertension 1998, 32: 10.
    • (1998) Hypertension , vol.32 , pp. 10
    • Blackwood, A.1    Baker, E.H.2    Dong, Y.B.3
  • 23
    • 0025648068 scopus 로고
    • Alterations in aldosterone secretion and metabolism in low renin hypertension
    • Griffing GT, Wilson TE, Melby JC: Alterations in aldosterone secretion and metabolism in low renin hypertension. J Clin Endocrinol Metab 1990, 71: 1454-1460.
    • (1990) J Clin Endocrinol Metab , vol.71 , pp. 1454-1460
    • Griffing, G.T.1    Wilson, T.E.2    Melby, J.C.3
  • 24
    • 0032886569 scopus 로고    scopus 로고
    • Genotype-phenotype relationships for the renin-angiotensin-aldosterone system in a normal population
    • Paillard F, Chansel D, Brand E, et al.: Genotype-phenotype relationships for the renin-angiotensin-aldosterone system in a normal population. Hypertension 1999, 34: 423-429.
    • (1999) Hypertension , vol.34 , pp. 423-429
    • Paillard, F.1    Chansel, D.2    Brand, E.3
  • 25
    • 0028144165 scopus 로고
    • Regulation of aldosterone secretion in hypertensive blacks
    • Fisher ND, Gleason RE, Moore TJ, et al.: Regulation of aldosterone secretion in hypertensive blacks. Hypertension 1994, 23: 179-184.
    • (1994) Hypertension , vol.23 , pp. 179-184
    • Fisher, N.D.1    Gleason, R.E.2    Moore, T.J.3
  • 26
    • 0033108030 scopus 로고    scopus 로고
    • The epithelial sodium channel in hypertension
    • Warnock DG: The epithelial sodium channel in hypertension. Curr Hypertens Rep 1999, 1: 158-163. Reports global suppression of urinary 18-hyrdroxylase metabolite excretion in patients with Liddle's syndrome.
    • (1999) Curr Hypertens Rep , vol.1 , pp. 158-163
    • Warnock, D.G.1
  • 27
    • 0027414682 scopus 로고
    • Mass spectrometry in the diagnosis of steroid-related disorders and in hypertension research
    • Shackleton CH: Mass spectrometry in the diagnosis of steroid-related disorders and in hypertension research. J Steroid Biochem Molec Biol 1993, 45: 127-140.
    • (1993) J Steroid Biochem Molec Biol , vol.45 , pp. 127-140
    • Shackleton, C.H.1
  • 28
    • 0032885747 scopus 로고    scopus 로고
    • Altered adrenal sensitivity to angiotensin II in low-renin essential hypertension
    • Fisher NDL, Hurwitz S, Ferri C, et al.: Altered adrenal sensitivity to angiotensin II in low-renin essential hypertension. Hypertension 1999, 34: 388-394. On closer examination, "nonmodulating" essential hypertension appears to have suppressed adrenal 18-hydroxylase activity related to chronic volume expansion despite "normal" nonsuppressed plasma renin activity.
    • (1999) Hypertension , vol.34 , pp. 388-394
    • Fisher, N.D.L.1    Hurwitz, S.2    Ferri, C.3
  • 29
    • 0031594450 scopus 로고    scopus 로고
    • Liddle syndrome: An autosomal dominant form of human hypertension
    • Warnock DG: Liddle syndrome: an autosomal dominant form of human hypertension. Kidney Int 1998, 53: 18-24.
    • (1998) Kidney Int , vol.53 , pp. 18-24
    • Warnock, D.G.1
  • 30
    • 0032887273 scopus 로고    scopus 로고
    • Low-renin and nonmodulating essential hypertension
    • Editorial comment on Fisher et al. 28
    • Warnock DG: Low-renin and nonmodulating essential hypertension. Hypertension 1999, 34: 395-397. Editorial comment on Fisher et al. [28].
    • (1999) Hypertension , vol.34 , pp. 395-397
    • Warnock, D.G.1
  • 31
    • 0031861245 scopus 로고    scopus 로고
    • Mutations in the mineralocorticoid receptor gene cause autosomal dominant pseudohypoaldosteronism, type I
    • Geller DS, Rodriquez-Soriano J, Boado AV, et al.: Mutations in the mineralocorticoid receptor gene cause autosomal dominant pseudohypoaldosteronism, type I. Nature Genet 1998, 19: 279-281.
    • (1998) Nature Genet , vol.19 , pp. 279-281
    • Geller, D.S.1    Rodriquez-Soriano, J.2    Boado, A.V.3
  • 32
    • 4243570933 scopus 로고    scopus 로고
    • A new Mendelian form of human hypertension caused by an activating mutation in the mineralocorticoid receptor [abstract]
    • Geller DS, Farhi A, Moritz M, et al.: A new Mendelian form of human hypertension caused by an activating mutation in the mineralocorticoid receptor [abstract]. J Am Soc Nephrol 1999, 10: 345A.
    • (1999) J Am Soc Nephrol , vol.10
    • Geller, D.S.1    Farhi, A.2    Moritz, M.3
  • 33
    • 0032878222 scopus 로고    scopus 로고
    • Levels of mineralocorticoids in whites and blacks
    • Pratt JH, Rebhun JF, Zhou L, et al.: Levels of mineralocorticoids in whites and blacks. Hypertension 1999, 34: 315-319.
    • (1999) Hypertension , vol.34 , pp. 315-319
    • Pratt, J.H.1    Rebhun, J.F.2    Zhou, L.3
  • 34
    • 0025729737 scopus 로고
    • Inappropriate elevation of the aldosterone/plasma renin activity ratio in hypertensive patients and 18-hydroxy-11-deoxycorticosterone: A subtype of essential hypertension?
    • Komiya I, Yamada T, Aizawa T, et al.: Inappropriate elevation of the aldosterone/plasma renin activity ratio in hypertensive patients and 18-hydroxy-11-deoxycorticosterone: a subtype of essential hypertension? Cardiology 1991, 78: 99-110.
    • (1991) Cardiology , vol.78 , pp. 99-110
    • Komiya, I.1    Yamada, T.2    Aizawa, T.3
  • 35
    • 0029160972 scopus 로고
    • Human hypertension caused by mutations in the kidney isozyme of 11 betahydroxysteroid dehydrogenase
    • Mune T, Rogerson FM, Nikkila H, et al.: Human hypertension caused by mutations in the kidney isozyme of 11 betahydroxysteroid dehydrogenase. Nature Genet 1995, 10: 394-399.
    • (1995) Nature Genet , vol.10 , pp. 394-399
    • Mune, T.1    Rogerson, F.M.2    Nikkila, H.3
  • 36
    • 0029060080 scopus 로고
    • A mutation in the HSD11B2 gene in a family with apparent mineralocorticoid excess
    • Wilson RC, Krozowski ZS, Li K, et al.: A mutation in the HSD11B2 gene in a family with apparent mineralocorticoid excess. J Clin Endocrinol Metab 1995, 80: 2263-2266.
    • (1995) J Clin Endocrinol Metab , vol.80 , pp. 2263-2266
    • Wilson, R.C.1    Krozowski, Z.S.2    Li, K.3
  • 37
    • 0023743171 scopus 로고
    • Mineralocorticoid action: Target tissue specificity is enzyme, not receptor mediated
    • Funder JW, Pearce PT, Smith AT: Mineralocorticoid action: target tissue specificity is enzyme, not receptor mediated. Science 1987, 242: 583-585.
    • (1987) Science , vol.242 , pp. 583-585
    • Funder, J.W.1    Pearce, P.T.2    Smith, A.T.3
  • 38
    • 0023242981 scopus 로고
    • Mineralocorticoid activity of liquorice: 11β-hydroxysteroid dehydrogenase deficiency comes of age
    • Stewart PM, Valentino R, Wallace AM, et al.: Mineralocorticoid activity of liquorice: 11β-hydroxysteroid dehydrogenase deficiency comes of age. Lancet 1987, 2: 821-824.
    • (1987) Lancet , vol.2 , pp. 821-824
    • Stewart, P.M.1    Valentino, R.2    Wallace, A.M.3
  • 39
    • 0030069526 scopus 로고    scopus 로고
    • Grapefruit juice and its flavonoids inhibit 11β-hydroxysteroid dehydrogenase
    • Lee YS, Lorenzo BJ, Koufis T, et al.: Grapefruit juice and its flavonoids inhibit 11β-hydroxysteroid dehydrogenase. Clin Pharmacol Ther 1996, 59: 62-71.
    • (1996) Clin Pharmacol Ther , vol.59 , pp. 62-71
    • Lee, Y.S.1    Lorenzo, B.J.2    Koufis, T.3
  • 40
    • 0030598951 scopus 로고    scopus 로고
    • Naringin and naringenin are not the primary CYP3A inhibitors in grapefruit juice
    • Edwards DJ, Bernier SM: Naringin and naringenin are not the primary CYP3A inhibitors in grapefruit juice. Life Sci 1996, 59: 1025-1030.
    • (1996) Life Sci , vol.59 , pp. 1025-1030
    • Edwards, D.J.1    Bernier, S.M.2
  • 41
    • 0029888213 scopus 로고    scopus 로고
    • Sodium status, corticosteroid metabolism and blood pressure in normal human subjects and in a patient with abnormal salt appetite
    • Ingram MC, Wallace AM, Collier A, et al.: Sodium status, corticosteroid metabolism and blood pressure in normal human subjects and in a patient with abnormal salt appetite. Clin Exp Pharmacol Physiol 1996, 23: 375-378.
    • (1996) Clin Exp Pharmacol Physiol , vol.23 , pp. 375-378
    • Ingram, M.C.1    Wallace, A.M.2    Collier, A.3
  • 42
    • 0028967443 scopus 로고
    • Evidence of coexisting changes in 11β-hydroxysteroid dehydrogenase and 5b reductase activity in subjects with untreated essential hypertension
    • Soro A, Ingram MC, Tonolo G, et al.: Evidence of coexisting changes in 11β-hydroxysteroid dehydrogenase and 5b reductase activity in subjects with untreated essential hypertension. Hypertension 1995, 25: 67-70.
    • (1995) Hypertension , vol.25 , pp. 67-70
    • Soro, A.1    Ingram, M.C.2    Tonolo, G.3
  • 43
    • 0027320739 scopus 로고
    • Deficient inactivation of cortisol by 11β-hydroxysteroid dehydrogenase in essential hypertension
    • Walker BR, Stewart PM, Shackleton CHL, et al.: Deficient inactivation of cortisol by 11β-hydroxysteroid dehydrogenase in essential hypertension. Clin Endocrinol 1993, 39: 221-227.
    • (1993) Clin Endocrinol , vol.39 , pp. 221-227
    • Walker, B.R.1    Stewart, P.M.2    Shackleton, C.H.L.3
  • 44
    • 0033305356 scopus 로고    scopus 로고
    • Molecular basis of human salt sensitivity: The role of the 11beta-hydroxysteroid dehydrogenase type 2
    • Lovati E, Ferrari P, Dick B, et al.: Molecular basis of human salt sensitivity: the role of the 11beta-hydroxysteroid dehydrogenase type 2. J Clin Endocrinol Metab 1999, 84: 3745-3749. Although 11β-hydroxy steroid dehydrogenase type II activity does not appear to be modulated by dietary salt intake, there may be an effect on systemic hypertension of dietary salt excess related to the 11β-hydroxy steroid dehydrogenase type II genotype.
    • (1999) J Clin Endocrinol Metab , vol.84 , pp. 3745-3749
    • Lovati, E.1    Ferrari, P.2    Dick, B.3
  • 45
    • 0347478279 scopus 로고    scopus 로고
    • Genetic association of 11 beta-hydroxysteroid dehydrogenase type 2(HSD11B2) flanking microsatellites with essential hypertension in blacks
    • Watson BJ, Bergman SM, Myracle A, et al.: Genetic association of 11 beta-hydroxysteroid dehydrogenase type 2(HSD11B2) flanking microsatellites with essential hypertension in blacks. Hypertension 1996, 28: 478-482.
    • (1996) Hypertension , vol.28 , pp. 478-482
    • Watson, B.J.1    Bergman, S.M.2    Myracle, A.3
  • 46
    • 0032506549 scopus 로고    scopus 로고
    • Cure of apparent mineralocorticoid excess by kidney transplantation
    • Palermo M, Cossu M, Shackleton CHL: Cure of apparent mineralocorticoid excess by kidney transplantation. N Engl J Med 1998, 339: 1787-1788. The apparent mineralocorticoid excess syndrome is a renal disorder, like Liddle's syndrome, that is "cured" by renal transplantation. Urinary excretion of the reduced metabolites of cortisol compared with cortisone may be misleading; measurements of urinary-free cortisol and free cortisone may be more directly relevant to the underlying pathophysiology.
    • (1998) N Engl J Med , vol.339 , pp. 1787-1788
    • Palermo, M.1    Cossu, M.2    Shackleton, C.H.L.3
  • 47
    • 0031723991 scopus 로고    scopus 로고
    • A new polymorphic restriction site in the human 11 beta-hydroxysteroid dehydrogenase type 2 gene
    • Smolenicka Z, Bach E, Schaer A, et al.: A new polymorphic restriction site in the human 11 beta-hydroxysteroid dehydrogenase type 2 gene. J Clin Endocrin Metabol 1998, 83: 1814-1817.
    • (1998) J Clin Endocrin Metabol , vol.83 , pp. 1814-1817
    • Smolenicka, Z.1    Bach, E.2    Schaer, A.3
  • 48
    • 0031740024 scopus 로고    scopus 로고
    • Importance of the renin system in determining blood pressure fall with salt restriction in black and white hypertensives
    • He FJ, Markandu ND, Sagnella GA, et al.: Importance of the renin system in determining blood pressure fall with salt restriction in black and white hypertensives. Hypertension 1998, 32: 820-824.
    • (1998) Hypertension , vol.32 , pp. 820-824
    • He, F.J.1    Markandu, N.D.2    Sagnella, G.A.3
  • 49
    • 0030020027 scopus 로고    scopus 로고
    • Liddle's disease: Abnormal regulation of amiloride-sensitive Na+ channels by b-subunit mutation
    • Bubien JK, Ismailov II, Berdiev BK, et al.: Liddle's disease: abnormal regulation of amiloride-sensitive Na+ channels by b-subunit mutation. Am J Physiol (Cell Physiol) 1996, 270: C208-C213.
    • (1996) Am J Physiol (Cell Physiol) , vol.270
    • Bubien, J.K.1    Ismailov, I.I.2    Berdiev, B.K.3
  • 50
    • 0032127970 scopus 로고    scopus 로고
    • Abnormalities of nasal potential difference measurement in Liddle's syndrome
    • Baker E, Jeunemaitre X, Portal AJ, et al.: Abnormalities of nasal potential difference measurement in Liddle's syndrome. J Clin Invest 1998, 102: 10-14.
    • (1998) J Clin Invest , vol.102 , pp. 10-14
    • Baker, E.1    Jeunemaitre, X.2    Portal, A.J.3
  • 51
    • 0032944361 scopus 로고    scopus 로고
    • Epithelial sodium channel activity is not increased in hypertension in whites
    • Baker EH, Portal AJ, McElvaney TA, et al.: Epithelial sodium channel activity is not increased in hypertension in whites. Hypertension 1999, 33: 1031-1035.
    • (1999) Hypertension , vol.33 , pp. 1031-1035
    • Baker, E.H.1    Portal, A.J.2    McElvaney, T.A.3
  • 52
    • 84900543431 scopus 로고    scopus 로고
    • Epithelial sodium channel-dependent hypertension: An emerging syndrome: Clinical and hormonal characteristics [abstract]
    • Gadallah MF, Mignone J, Habashi K, et al.: Epithelial sodium channel-dependent hypertension: an emerging syndrome: clinical and hormonal characteristics [abstract]. J Am Soc Nephrol 1999, 10: 365A.
    • (1999) J Am Soc Nephrol , vol.10
    • Gadallah, M.F.1    Mignone, J.2    Habashi, K.3
  • 53
    • 0030068024 scopus 로고    scopus 로고
    • Molecular genetics of human blood pressure variation
    • Lifton RP: Molecular genetics of human blood pressure variation. Science 1996, 272: 676-680.
    • (1996) Science , vol.272 , pp. 676-680
    • Lifton, R.P.1


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